State-of-the-Art Review : Secondary Prevention of Myocardial Infarction: Beneficial Effect of Combining Oral Anticoagulant Plus Aspirin

Altman, Raúl; Rouvier, Jorge; Scazziota, Alejandra

doi:10.1177/107602960000600302

Cited by 4 publications

(2 citation statements)

References 68 publications

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“…With the focus on coronary occlusive disease, there are other origins of thrombotic events beside plaque rupture [15,16]. In atheroma the luminal surface is irregular and sometimes eroded, and the lack of endothelial cells constitutes a vulnerable site, as prone to acute thrombosis as lipid-rich plaques are [17].…”

Section: Reviewmentioning

confidence: 99%

Risk factors in coronary atherosclerosis athero-inflammation: the meeting point

Altman

2003

Thrombosis J

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Section: Reviewmentioning

confidence: 99%

Risk factors in coronary atherosclerosis athero-inflammation: the meeting point

Altman

2003

Thrombosis J

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“…In 1972, Sinapius reported that in a necropsy of 91 patients who died of myocardial infarction, 45% presented a red thrombus, 15% a platelet thrombus, and 40% a mixed thrombus (6). More recently, and according to angioscopies performed 30 days after acute myocardial infarction (AMI), Van Belle and associates found that 12% of the patients undergoing thrombolysis and 30% of those undergoing no thrombolysis presented white (platelet) thrombi, 24% and 30% red thrombi, and 64% and 40% mixed thrombi, respectively (7).…”

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confidence: 99%

Facilitated Thrombolysis: Dethrombosis?

Luciardi

Berman²,

Muntaner³

et al. 2002

Clin Appl Thromb Hemost

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Intracoronary thrombus formation results from the combined effects of platelet aggregation and fibrin formation. Fibrinogen plays a significant role in each of these components. Dethrombosis is a therapeutic approach that allows dissolution of a recent thrombus while avoiding the potent fibrinolytic therapies. The less aggressive strategies suggested to achieve dethrombosis are antifibrin, antiplatelet, and defibrinogenation. Glycoprotein receptor antagonists have been beneficial in patients undergoing percutaneous transluminal coronary angioplasty; however, little is known about their potency of inducing reperfusion in acute myocardial infarction. Early use of the strategies suggested to induce dethrombosis (antifibrin, antiplatelet, and defibrinogenation) may facilitate fibrinolysis and primary angioplasty and further induce reperfusion. Nevertheless, the theoretical arguments of facilitated thrombolysis (dethrombosis) have not yet been fruitful as a major clinical benefit except in patients undergoing primary percutaneous coronary intervention.

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Medical Management of Coronary Artery Disease

Farag

Zamvar

2015

PanVascular Medicine

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State-of-the-Art Review : Secondary Prevention of Myocardial Infarction: Beneficial Effect of Combining Oral Anticoagulant Plus Aspirin

Cited by 4 publications

References 68 publications

Risk factors in coronary atherosclerosis athero-inflammation: the meeting point

Risk factors in coronary atherosclerosis athero-inflammation: the meeting point

Facilitated Thrombolysis: Dethrombosis?

Medical Management of Coronary Artery Disease

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