2000
DOI: 10.1016/s0024-3205(99)00663-3
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Stimulatory effect of trans-cinnamaldehyde on noradrenaline secretion in guinea-pig ileum myenteric nerve terminals

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Cited by 9 publications
(3 citation statements)
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“…The result found that berberine can improve oxidative stress and cholinesterase activity, berberine has improved memory dysfunction caused by abnormal oxidative stress and cholinergic function. Cinnamaldehyde, a main component of traditional Chinese medicine cinnamon, is biologically active as anti-inflammatory and anti-oxidative agent [ 51 , 52 ], evidenced by inhibited the activity of inflammatory factors NF-kb, Nosynthase, COX-2 [ 53 ] and suppressed release of noradernaline in guinea-pig ileum myenteric nerve terminals [ 54 , 55 ]. In the experiment, Yang D [ 19 ] found cinnamaldehyde have anti-oxidation and anti-inflammatory effects, and it had repair and protection effects on the injured neurons of rat dorsal ganglion.…”
Section: Discussionmentioning
confidence: 99%
“…The result found that berberine can improve oxidative stress and cholinesterase activity, berberine has improved memory dysfunction caused by abnormal oxidative stress and cholinergic function. Cinnamaldehyde, a main component of traditional Chinese medicine cinnamon, is biologically active as anti-inflammatory and anti-oxidative agent [ 51 , 52 ], evidenced by inhibited the activity of inflammatory factors NF-kb, Nosynthase, COX-2 [ 53 ] and suppressed release of noradernaline in guinea-pig ileum myenteric nerve terminals [ 54 , 55 ]. In the experiment, Yang D [ 19 ] found cinnamaldehyde have anti-oxidation and anti-inflammatory effects, and it had repair and protection effects on the injured neurons of rat dorsal ganglion.…”
Section: Discussionmentioning
confidence: 99%
“…Release of noradrenaline by paeoni¯orin from noradrenergic nerve terminals was characterized in two ways. One was blockade with guanethidine at concentrations su cient to block noradrenergic nerve terminals (Cheng et al 2000). Guanethidine has been documented as a speci® c blocker of noradrenergic nerve terminals (Starke 1972).…”
Section: Discussionmentioning
confidence: 99%
“…Állatkísérletekben többek között igazolták, hogy a fahéjaldehid gátolja a Na + -K + -ATP-ázt a jejunumban [10], stimulálja a noradrenalin szekrécióját az ileum myentericus idegvégződéseiben [11]. A fahéjaldehid gátolja a NO-szintézist és szuppresszálja az indukálható NO-szintáz (iNOS) expresszióját RAW 246.7 makrofágkultúrában lipopoliszacharid-indukció alatt, valamint mind a fahéjaldehid, mind a 2'-metoxi, illetve 2'-hidroxi származékai gátolják a nukleárisfaktor-kappa-B (NF-κB) aktivációját [12,13].…”
Section: Mikrobiológiai Biokémiai éS Molekuláris Biológiai Kutatásokunclassified