“…Godil [16] and coworkers compared the H. pylori prevalence in stress ulcer bleeding with the prevalence in peptic ulcer bleeding and found a lower prevalence in the group of stress ulcer bleeding, although the patients in this group were older than in our study. The reason for this low prevalence is not clear, but it can be assumed that the H. pylori induced production of prostaglandin E2, which is responsible for a protective effect against stress lesions of the gastric mucosa.…”
Our results suggest that the Helicobacter pylori infection does not play a pathogenic role in stress ulcer bleeding. Prophylactic cure of Helicobacter pylori can not be recommended in this setting.
“…Godil [16] and coworkers compared the H. pylori prevalence in stress ulcer bleeding with the prevalence in peptic ulcer bleeding and found a lower prevalence in the group of stress ulcer bleeding, although the patients in this group were older than in our study. The reason for this low prevalence is not clear, but it can be assumed that the H. pylori induced production of prostaglandin E2, which is responsible for a protective effect against stress lesions of the gastric mucosa.…”
Our results suggest that the Helicobacter pylori infection does not play a pathogenic role in stress ulcer bleeding. Prophylactic cure of Helicobacter pylori can not be recommended in this setting.
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