Surgical outcome of oculomotor nerve palsy in pituitary adenoma

Chuang, Chi‐Cheng; Chen, E.; Huang, Yin-Cheng; Tu, Po‐Hsun; Chen, Yao-Liang; Pai, Ping‐Ching

doi:10.1016/j.jocn.2011.02.041

Cited by 17 publications

(16 citation statements)

References 26 publications

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“…The postoperative complete recovery of the affected oculomotor nerve by pituitary tumor is reported to occur in 68% to 100% . However, both cases in the present series exhibited only partial recovery.…”

Section: Discussioncontrasting

confidence: 51%

“…The postoperative complete recovery of the affected oculomotor nerve by pituitary tumor is reported to occur in 68% to 100%. 14,[16][17][18] However, both cases in the present series exhibited only partial recovery. Compared with the previous reports, of which the major proportion consisted of apoplectic patients with the sudden onset of symptoms, both cases of the present series was nonapoplectic and required considerable time until the treatment decision was made by the patient due to the slow progressive nature of the symptoms.…”

Section: Prognosis Of Oculomotor Nerve Palsycontrasting

confidence: 57%

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Endoscopic endonasal surgery for pituitary adenomas extending to the oculomotor cistern

et al. 2017

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Section: Discussioncontrasting

confidence: 51%

Section: Prognosis Of Oculomotor Nerve Palsycontrasting

confidence: 57%

Endoscopic endonasal surgery for pituitary adenomas extending to the oculomotor cistern

et al. 2017

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“…Other than early treatment, Chuang et al, in 2011, also suggested that absence of afferent pupillary defect and low-intensity ocular motor signs are factors of good ocular motor recovery [48].…”

Section: Pituitary Apoplexymentioning

confidence: 99%

Neuro-ophthalmologic exploration in non-functioning pituitary adenoma

Abouaf

Vighetto

Lebas³

2015

Annales d'Endocrinologie

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Non-functioning pituitary adenoma may lead to blindness and causes visual impairment in 58% of cases and, more rarely, ocular motor disorder. Patients are slow to become aware of their visual dysfunction, vision in one eye compensating the deficit in the other. Assessment of visual function, comprising visual acuity and visual field evaluation and fundus examination, should be performed regularly according to the severity of impairment. Optic nerve optical coherence tomography (OCT) can quantify optic atrophy reproducibly, and is of prognostic value for postoperative visual recovery. Diplopia most often involves decompensation of heterophoria, visual field fusion being hampered by the visual field defect; such diplopia without ocular motor deficit is known as "hemifield slide". Diplopia associated with ocular motor palsy is caused by tumoral invasion of the cavernous sinus (IIIrd, IVth or VIth nerve palsy); in large impairment, restricted eye movement is easily observed; milder palsies require neuro-ophthalmologic assessment and/or Lancaster test. Pituitary apoplexy induces ocular motor impairment in 70% of cases, strongly guiding diagnosis. Visual impairment is associated in 75% of cases. The degree of neuro-ophthalmologic (visual and ocular motor) impairment is one of the main criteria guiding treatment of pituitary apoplexy (conservative medical and/or surgical treatment) and follow-up.

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“…I apologized I did not mention the explanation about consciousness disturbance in patients in group C. Consciousness disturbance occurred in 3 patients in our study, and all of them were included only in group C (Page 13, line [1][2][3]. We believe that their consciousness disturbance was caused by pituitary hormonal insufficiency (hypopituitarism), because their consciouness disturbance improved remarkably soon after administration of corticosteroid.…”

mentioning

confidence: 72%