Survival after severe intrathoracic electrical injury

Schleich, Arno R.; Schweiger, H.; Becsey, A.; Cruse, C. Wayne

doi:10.1016/j.burns.2009.06.207

Cited by 13 publications

(15 citation statements)

References 15 publications

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“…Los casos presentador por Masanes MJ. y co y por Schleich A. y col. fueron trabajadores de compañías eléctricas que estuvieron expuestos a corriente eléctrica de alta tensión y presentaron consolidación focal en la imagen pulmonar, con requerimiento de resección quir úrgica (7,8) . La lesión focal del pulmón también se ha descrito en un empleado después de exposición a energía de baja tensión (380 voltios) complicado con paro cardiorespiratorio y edema cerebral (9) .…”

Section: Discussionunclassified

Lesión pulmonar por trauma eléctrico

Gemelli

Bisso

Crusat

et al. 2020

Rev Fac Cien Med Univ Nac Cordoba

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Introducción: Las quemaduras eléctricas representan entre el 3 y el 4% de todas las quemaduras. Los sitios de impacto más frecuentes son el sistema cardiovascular, el tejido muscular, el neurológico y el cutáneo. El compromiso del sistema respiratorio es infrecuente, con escaso número de casos reportados en la literatura. Caso clinico: Paciente varón de 26 años que ingresa luego de lesión eléctrica de alto voltaje. Presentó compromiso cutáneo y distress respiratorio con requerimiento de asistencia respiratoria mecánica, sedación profunda y bloqueo neuromuscular. Conclusión: Hay pocos casos reportados en la literatura de lesión pulmonar asociado a trauma eléctrico. Es fundamental reconocer el sistema respiratorio como posible sitio de impacto resaltando la importancia de un soporte vital avanzado.

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Section: Discussionunclassified

Lesión pulmonar por trauma eléctrico

Gemelli

Bisso

Crusat

et al. 2020

Rev Fac Cien Med Univ Nac Cordoba

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“…Two subsequent cases in electrical workers were due to the high‐voltage electric current (≥1000 V), had focal consolidation on lung imaging, and were managed with surgical resection. Histopathological findings included lung infarction, coagulative necrosis, and lipid pneumopathy in one case on Day 4 and infarction, liquefaction, and abscess formation in the other on Day 27 . Focal lung injury has also been reported in an electrical worker after a low‐voltage shock (380 V) complicated by respiratory arrest in the field and subsequent cerebral oedema.…”

Section: Discussionmentioning

confidence: 99%

“…To our knowledge, there have been four English‐language case reports of electricity‐associated lung damage . The first case was an episode of acute cardiogenic pulmonary oedema that followed electricity‐induced ventricular fibrillation and cardiopulmonary resuscitation, with documentation of high pulmonary artery occlusion pressure, low cardiac index, and low oxygen saturation of mixed venous blood .…”

Section: Discussionmentioning

confidence: 99%

“…In the only example of pulmonary oedema due to electrical injury in the literature, arrhythmia leading to cardiac arrest was considered to be the cause . In other reported cases, the electric current appears to directly injure lung tissue, with focal consolidation on radiographs and necrosis and coagulation on histopathological examination . The electrical injury may lead to additional complications, such as haemoptysis, acute respiratory distress, and infection.…”

Section: Introductionmentioning

confidence: 99%

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Low‐voltage electricity‐induced lung injury

Truong

Smith

et al. 2017

Respirology Case Reports

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We report a case of bilateral pulmonary infiltrates and haemoptysis following low‐voltage electricity exposure in an agricultural worker. A 58‐year‐old man standing in water reached for an electric watering machine and sustained an exposure to 220 V circuit for an uncertain duration. The electricity was turned off by another worker, and the patient was asymptomatic for the next 10 h until he developed haemoptysis. A chest radiograph demonstrated bilateral infiltrates, and chest computed tomography (CT) revealed ground‐glass opacities with interstitial thickening. Evaluations, including electrocardiogram, serum troponin, N‐terminal pro‐B‐type natriuretic peptide (NT‐pro BNP), coagulation studies, and echocardiogram, found no abnormality. The patient was treated for suspected electricity‐induced lung injury and bleeding with tranexamic acid and for rhabdomyolysis with volume resuscitation. He recovered with complete resolution of chest radiograph abnormalities by Day 7. This is the first reported case of bilateral lung oedema and/or injury after electricity exposure without cardiac arrest.

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“…Thus, the digestive tract mucosa is injured. Whether the application of anticoagulants at an early stage can protect the digestive tract mucosa and reduce the gastrointestinal reaction remains to be elucidated ( 15 ). In the present study, it was demonstrated that early heparin + antithrombin treatment significantly inhibited the lung injury induced by severe burns complicated by inhalation injury in rabbits, and reduced the levels of PaO 2 , PaCO 2 and SPO 2 .…”

Section: Discussionmentioning

confidence: 99%

Early anticoagulation therapy for severe burns complicated by inhalation injury in a rabbit model

Guo

Xiong

et al. 2017

Molecular Medicine Reports

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The aim of the present study was to determine the effects of early anticoagulation treatment on severe burns complicated by inhalation injury in a rabbit model. Under anesthetization, an electrical burns instrument (100°C) was used to scald the backs of rabbits for 15 sec, which established a 30% III severe burns model. Treatment of the rabbits with early anticoagulation effectively improved the severe burns complicated by inhalation injury-induced lung injury, reduced PaO2, PaCO2 and SPO2 levels, suppressed the expression of tumor necrosis factor-α, interleukin (IL)-1β and IL-6, and increased the activity of IL-10. In addition, it was found that early anticoagulation treatment effectively suppressed the activities of caspase-3 and caspase-9, upregulated the protein expression of vascular endothelial growth factor (VEGF) and decreased the protein expression of protease-activated receptor 1 (PAR1) in the severe burns model. It was concluded that early anticoagulation treatment affected the severe burns complicated by inhalation injury in a rabbit model through the upregulation of VEGF and downregulation of PAR1 signaling pathways. Thus, early anticoagulation is a potential therapeutic option for severe burns complicated by inhalation injury.

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Survival after severe intrathoracic electrical injury

Cited by 13 publications

References 15 publications

Lesión pulmonar por trauma eléctrico

Lesión pulmonar por trauma eléctrico

Low‐voltage electricity‐induced lung injury

Early anticoagulation therapy for severe burns complicated by inhalation injury in a rabbit model

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