Task force I: Standardization of terminology and interpretation

Surawicz, Borys; Uhley, Herman N.; Borun, Raymond; Laks, Michael M.; Crevasse, Lamar; Rosen, Kenneth M.; Nelson, William E.; Mandel, William J.; Lawrence, Patrick; Jackson, L. K.; Flowers, Nancy C.; Clifton, James; Greenfield, Joseph C.; Medina, Etienne O.Rubles De

doi:10.1016/0002-9149(78)90147-9

Cited by 151 publications

(34 citation statements)

References 123 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…ECG diagnoses of abnormal T-wave morphology (either nonspecific T-wave abnormality, peaked T waves, or ST elevation), abnormal impulse generation (sinus bradycardia < 50 beats ⁄ minute, sinus arrest, or wide complex tachycardia > 100 beats ⁄ minute), and abnormal impulse conduction (first-degree atrioventricular block > 200 msec and either nonspecific interventricular conduction delay or bundle branch block > 120 msec), based on standardized cardiology consensus definitions for terminology and interpretation published by the American College of Cardiology Task Force. 26 The World Health Organization Task Force criteria for conduction abnormalities was used for differentiating right or left bundle branch from nonspecific interventricular conduction delay. 27 We used standard cardiology morphology criteria for defining the characteristic T-wave morphology of hyperkalemia, referred to in this study as ''peaked T waves.''…”

Section: Methods Of Measurementmentioning

confidence: 99%

Effects of Presentation and Electrocardiogram on Time to Treatment of Hyperkalemia

Freeman

Feldman

Mitchell

et al. 2008

Academic Emergency Medicine

View full text Add to dashboard Cite

Objectives: To assess the time to treatment for emergency department (ED) patients with critical hyperkalemia and to determine whether the timing of treatment was associated with clinical characteristics or electrocardiographic abnormalities. Methods:The authors performed a retrospective chart review of ED patients with the laboratory diagnosis of hyperkalemia (potassium level > 6.0 mmol ⁄ L). Patients presenting in cardiac arrest or who were referred for hyperkalemia or dialysis were excluded. Patient charts were reviewed to find whether patients received specific treatment for hyperkalemia and, if so, what clinical attributes were associated with the time to initiation of treatment.Results: Of 175 ED visits that occurred over a 1-year time period, 168 (96%) received specific treatment for hyperkalemia. The median time from triage to initiation of treatment was 117 minutes (interquartile range [IQR] = 59 to 196 minutes). The 7 cases in which hyperkalemia was not treated include 4 cases in which the patient was discharged home, with a missed diagnosis of hyperkalemia. Despite initiation of specific therapy for hyperkalemia in 168 cases, 2 patients died of cardiac arrhythmias. Among the patients who received treatment, 15% had a documented systolic blood pressure (sBP) < 90 mmHg, and 30% of treated patients were admitted to intensive care units. The median potassium value was 6.5 mmol ⁄ L (IQR = 6.3 to 7.1 mmol ⁄ L). The predominant complaints were dyspnea (20%) and weakness (19%). Thirty-six percent of patients were taking angiotensin-converting enzyme (ACE) inhibitors. Initial electrocardiograms (ECGs) were abnormal in 83% of patient visits, including 24% of ECGs with nonspecific ST abnormalities. Findings of peaked T-wave morphology (34%), first-degree atrioventricular block (17%), and interventricular conduction delay (12%) did not lead to early treatment. Vital sign abnormalities, including hypotension (sBP < 90 mmHg), were not associated with early treatment. The chief complaint of ''unresponsive'' was most likely to lead to early treatment; treatment delays occurred in patients not transported by ambulance, those with a chief complaint of syncope and those with a history of hypertension.Conclusions: Recognition of patients with severe hyperkalemia is challenging, and the initiation of appropriate therapy for this disorder is frequently delayed.ACADEMIC EMERGENCY MEDICINE 2008; 15:239-249 ª

show abstract

Section: Methods Of Measurementmentioning

confidence: 99%

Effects of Presentation and Electrocardiogram on Time to Treatment of Hyperkalemia

Freeman

Feldman

Mitchell

et al. 2008

Academic Emergency Medicine

View full text Add to dashboard Cite

show abstract

“…Tall P waves with the amplitude ≧0.25 mV in inferior leads have been regarded as an ECG sign representing RA overload (RAO). 39 Typically, patients with COPD exhibit vertical P-wave axis with peaked P wave in leads II, III, and aVF (P pulmonale) (Figure 2). 40,41 Fig.…”

Section: P Pulmonale and Afmentioning

confidence: 99%

Prognostic Value of P Wave for Developing Atrial Fibrillation

Hayashi¹,

Horie²

2012

Atrial Fibrillation - Basic Research and Clinical Applications

View full text Add to dashboard Cite

“…19,20 Based on pathological correlation, a relationship between the location of infarcted areas and Q waves was accepted and, with minor modifications, implemented in scientific statements and textbooks. 21 According to these studies, Q waves correspond to the following myocardial segments: leads V 1 -V 2 to the septal wall, leads V 3 -V 4 to the anterior wall, leads I and aVL to the lateral wall, and leads II, III, and aVF to the inferior wall. The mirror pattern (high R wave) in leads V 1 -V 2 was traditionally considered as corresponding to the basal part of the inferoposterior wall (named true posterior MI).…”

Section: Association Between Segmental MI Ecg Signs Of Necrosismentioning

confidence: 99%

Correlation of Electrocardiogram and Regional Cardiac Magnetic Resonance Imaging Findings in ST‐Elevation Myocardial Infarction: A Literature Review

Rinta-Kiikka

Tuohinen

Ryymin

et al. 2014

Noninvasive Electrocardiol

View full text Add to dashboard Cite

Background:Patients with acute ST-elevation myocardial infarction (STEMI) benefit substantially from emergent coronary reperfusion. The principal mechanism is to open the occluded coronary artery to minimize myocardial injury. Thus the size of the area at risk is a critical determinant of the patient outcome, although other factors, such as reperfusion injury, have major impact on the final infarct size. Acute coronary occlusion almost immediately induces metabolic changes within the myocardium, which can be assessed with both the electrocardiogram (ECG) and cardiac magnetic resonance (CMR) imaging.Methods: The 12-lead ECG is the principal diagnostic method to detect and risk-stratify acute STEMI. However, to achieve a correct diagnosis, it is paramount to compare different ECG parameters with golden standards in imaging, such as CMR. In this review, we discuss aspects of ECG and CMR in the assessment of acute regional ischemic changes in the myocardium using the 17 segment model of the left ventricle presented by American Heart Association (AHA), and their relation to coronary artery anatomy.Results: Using the 17 segment model of AHA, the segments 12 and 16 remain controversial. There is an important overlap in myocardial blood supply at the antero-lateral region between LAD and LCx territories concerning these two segments.Conclusion: No all-encompassing correlation can be found between ECG and CMR findings in acute ischemia with respect to coronary anatomy.

show abstract

Task force I: Standardization of terminology and interpretation

Cited by 151 publications

References 123 publications

Effects of Presentation and Electrocardiogram on Time to Treatment of Hyperkalemia

Effects of Presentation and Electrocardiogram on Time to Treatment of Hyperkalemia

Prognostic Value of P Wave for Developing Atrial Fibrillation

Correlation of Electrocardiogram and Regional Cardiac Magnetic Resonance Imaging Findings in ST‐Elevation Myocardial Infarction: A Literature Review

Contact Info

Product

Resources

About