Test of h1(1830) made of $$K^* \bar K^*$$ with the $$\eta _c \to \varphi K^* \bar K^*$$ decay

Abstract

We present a new reaction, complementary to the J/ψ → ηK * 0K * 0 from which an h 1 resonance with mass around 1830 MeV was reported from a BESIII experiment. The new reaction is η c → φK * K * , or η c (2S) → φK * K * . Using the information from the analysis of J/ψ → ηK * 0K * 0 , we find that the K * K * invariant mass distribution for those two η c decays exhibits a clear peak around 1830 MeV perfectly distinguishable from what one obtains with pure phase space. We suggest the implementation of these reactions to assert the existence of this elusive resonance which, by its nature as a vector-vector molecule with 0 − (1 +− ) quantum numbers, only couples to the K * K * channel.

Rotation Tunable Photocatalytic Properties of ZnO/GaN Heterostructures

“…Combining two different semiconductors to construct a heterostructure is an effective strategy for enhancing photocatalytic performance, [17][18][19] which should be attributed to the fact that the heterostructure usually has smaller bandgap than the single semiconductor, and the formation of the built-in electric field in the heterostructure interface area can hinder photogenerated carriers. A series of heterostructures, such as C 2 N/ InTe, [20] AlN/WS 2 , [21] MoTe 2 /CrS 2 , [22] GaN/Mg(OH) 2 , [23] MoS 2 / CdS, [24] CdS/C 2 N, [25] CdS/ZnSe, [26] BlueP/Mg(OH) 2 , [27] BiNbO 4 /ZnWO 4 , [28] C 60 /g-C 3 N 4 , [29] SiC/MoS 2 , [30] and GaN/ BlueP, [31] exhibit enhanced photocatalytic performance. Especially, some ZnO monolayer-based heterostructures are ZnO/MoS 2 , [32,33] ZnO/WS 2 , [34] ZnO/WSe 2 , [35] ZnO/AlN, [36] and N-ZnO/g-C 3 N 4 .…”

The Development of a Macromolecular Analgesic for Arthritic Pain

“…It has long since been established the ability of HPMA copolymers to extravasate into solid tumor tissue via the enhanced permeability and retention (EPR) effect, due to the polymer's uncharged and flexible nature which allows passage through endothelial tissue layers. 35 Analogous to tumor targeting, our previous research [21][22][23][24] has proven the ability of HPMA copolymers to passively and selectively target inflamed tissue through what we have coined as the ELVIS mechanism; i.e. Extravasation through Leaky Vasculature and Inflammatory cell-mediated Sequestration.…”

“…20 On the other hand, macromolecular prodrugs based upon HPMA copolymers have been found to passively target inflammation in several animal models of human diseases. [21][22][23][24] This novel inflammation-targeting mechanism, termed ELVIS (Extravasation through Leaky Vasculature and Inflammatory cell-mediated Sequestration), can be utilized to selectively localize and retain therapeutic agents at the inflammatory sites. Given the ELVIS mechanism, which would facilitate the passive targeting of HPMA copolymers to the arthritic joints, and the impermeability of the BBB to water-soluble polymers, we hypothesized that upon systemic administration of an opioid-conjugated HPMA copolymer prodrug system in a rat model of monoarticular adjuvant-induced arthritis (MAA), a standard model often used in pain and inflammation research, the prodrug can preferentially extravasate through vascular leakage present in the arthritic joints and be sequestered by residential and infiltrated inflammatory cells that have been activated by inflammatory cytokines and possess high rates of endocytosis.…”

Signature of an $h_1$ state from $J/ψ\to ηK^{*0}\bar{K}^{*0}$ and theoretical description of the $Z_c(3900)$ and $Z_c(4020)$ as $D \bar D^*$ and $D^* \bar D^*$ molecular states

“…We think that these data offer enough evidence for this new state. In [4] we have made predictions for two more reactions η c → φK * K * , or η c (2S ) → φK * K * , that should help establish this resonance on firmer grounds. In Fig.…”