The Athletic Heart Syndrome

Gott, Peter H.

doi:10.1001/archinte.1968.00300090050011

Cited by 31 publications

(3 citation statements)

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“…1, 3, 8), mostly in V2-V 4, sometimes together with coving of the S-T segment is quite common (table I), its reported incidence is generally 10-30% [3,26,28], Negative T waves were reported in athletes by other authors [12,18,28], but are quite uncommon (table I). Generally they are not considered as a pathological finding [20,24], but some authors believe that they may be indicative of the condition of over training [6,8], Figure 8 shows negative T waves in leads II, III, AVF in a 17-year-old basketball player who was diagnosed as suffering from over training; however, the ECG pattern remained unaltered following a month's rest.…”

Section: Discussionmentioning

confidence: 99%

“…It is possible that the ST-T changes represent neurovegetative or metabolic disorders. The possibility of hypokalemia has also been mentioned [8]. Another possibility is that these changes manifest toxic myocardial damage, possibly due to intercurrent diseases that generally do not hinder the athletes from continuation of intensive training.…”

Section: Figmentioning

confidence: 99%

“…Another possibility is that these changes manifest toxic myocardial damage, possibly due to intercurrent diseases that generally do not hinder the athletes from continuation of intensive training. Some authors believe that the T wave changes may represent overtraining [6,8].…”

Section: Figmentioning

confidence: 99%

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Common ECG Changes in Athletes

Hanne-Paparo¹,

Drory²,

Schoenfeld³

et al. 1976

Cardiology

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The various ECG changes found in a group of 3,000 healthy athletes are discussed on the basis of eight representative ECG recordings. The common findings were sinus bradycardia, atrioventricular conduction disturbances, left and right ventricular hypertrophy according to the accepted voltage criteria, right axis deviation, intraventricular conduction disturbances (mainly in the right side of the heart), and various disturbances of the repolarization phase. The literature on this subject is reviewed, and the possible mechanisms involved in production of the various ECG changes are discussed. It is pointed out that in the absence of other evidence suggestive of cardiovascular disease, these changes should be considered as a normal variant and not lead to restriction of physical activity.

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