2000
DOI: 10.1016/s0021-9150(00)80158-6
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The effect of fluvastatin on catheter-induced intimal thickening in normocholesterolemic rabbits

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Cited by 4 publications
(11 citation statements)
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“…These results were subsequently reproduced by other workers using fluvastatin (8 mg/kg/day, po) [46] or simvastatin (2 or 20 mg/kg/day, sc) [49] in various models of catheter-induced vascular injury. More recently, Nishimura et al reported similar findings with simvastatin (2 mg/kg/day, po) in a rat model of chemically induced pulmonary artery hypertension [44].…”
Section: From Bench To Body: Clinical Relevance Of Statininduced Apopsupporting
confidence: 63%
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“…These results were subsequently reproduced by other workers using fluvastatin (8 mg/kg/day, po) [46] or simvastatin (2 or 20 mg/kg/day, sc) [49] in various models of catheter-induced vascular injury. More recently, Nishimura et al reported similar findings with simvastatin (2 mg/kg/day, po) in a rat model of chemically induced pulmonary artery hypertension [44].…”
Section: From Bench To Body: Clinical Relevance Of Statininduced Apopsupporting
confidence: 63%
“…H-ras suppression and p53 induction have also been documented upon in vivo statin treatment [46], where downstream effectors of the classical MAPK (c-fos) and p38 / JNK (c-jun) signalling pathways are also downregulated [44]. Taken together these observations suggest that statins may also directly intercept growth factor receptor signalling, probably at the level of Ras (Fig.…”
Section: Statins and The Cell Cyclementioning
confidence: 72%
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