The influence of drugs on disseminated intravascular coagulation (DIC) III. Effects of busulfan-induced thrombocytopenia and inhibition of platelet function by acetylsalicylic acid

Markwardt, Fritz; Nowak, G; Glusa, Erika; Hoffmann, A.

doi:10.1016/0049-3848(79)90054-9

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Bacterial-induced Cellular Activation Of Coagulation1

Injury and Bleeding Models1

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1980

2013

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Cited by 8 publications

(2 citation statements)

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“…Platelets are not necessary for DIC to occur in animal models of bacterial-induced DIC. 106,122,141 However, platelets may play an augmentatory role. 12,51,59,62,94,143,175,200,201 Platelets have binding sites for bacteria or bacterial constituents, such as endotoxin.…”

Section: Bacterial-induced Cellular Activation Of Coagulationmentioning

confidence: 99%

Infection-Induced Thrombocytopenia

Wilson¹,

Neame²,

Kelton³

1982

Semin Thromb Hemost

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Section: Bacterial-induced Cellular Activation Of Coagulationmentioning

confidence: 99%

Infection-Induced Thrombocytopenia

Wilson¹,

Neame²,

Kelton³

1982

Semin Thromb Hemost

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“…There are well-established protocols for inducing thrombocytopenia either chemically (busulfan) 66 , radiation-therapy, or repeat blood draws/transfusion, and have been tested in conjunction with other synthetic platelet treatments. 11,21,22,61 …”

Section: Injury and Bleeding Modelsmentioning

confidence: 99%