The Prevalence of Diet-Induced Migraine

Peatfield, R C; Glover, Vivette; Littlewood, JT; Sandler, M.; Fc, Rose

doi:10.1046/j.1468-2982.1984.0403179.x

Cited by 95 publications

(77 citation statements)

References 11 publications

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“…Anyhow, the figures in Table 5, the code 8.1.4 part, are heterogeneous. The figures from the English study [9,10] and the present one are probably rather far from being directly comparable. The first ones concern the frequency of "dietary migraine" inclusive of alcohol ingestion, in a hospital setting, i.e., in "established migraineurs".…”

Section: Discussioncontrasting

confidence: 43%

“…There seems to be a considerable scatter as regards frequency of alcohol-induced headache in the literature (code: 8.1.4, IHS) [7,9,10, present study] (Table 5). In an English study, a relatively high percentage did "report precipitation of headache by alcoholic drinks" [10].…”

Section: Discussionmentioning

confidence: 85%

“…Portugal, which has this pattern, there was a prevalence of headache due to alcohol of 0.3% [7] (Table 5). Porto study [7] 0.3 -Copenhagen study [8] -72 Peatfield et al [9,10] 29* Present study (a) 3; (b) 55** 64 Kaivola et al [6] Invariably*** *It is stated that foods (e.g., alcohol) precipitated headache [9, 10] **(a) Prevalence: a total of 34 out of 1122 (whole study group); (b) 55%=frequency among those with adequate exposure ***Selected trial persons (n=30), see text…”

Section: Discussionmentioning

confidence: 99%

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Hangover headache: various manifestations and proposal for criteria. Vågå study of headache epidemiology

Sjaastad

Bakketeig

2004

J Headache Pain

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IntroductionHangover headache is a well-known and characteristic manifestation of alcohol consumption. It appears in the wake of a drinking bout, when the blood alcohol concentration is falling (IHS code 8.3.1 [1]). There are few detailed descriptions of the panorama of manifestations of hangover headache in headache literature. The complaints during attack mainly seem to pertain to two major symptom categories: headache and gastro-intestinal (g-i) upset. The g-i discomfort in hangover headache in Vågå has been described elsewhere [2].In the present communication, particular attention will be paid to localisation, duration and intensity of the headache. The connection between red wine and headache will also be touched upon. The symptomatology of hangover headache in the Vågå study will be compared with the description given by Raskin [3]. Finally, we will propose criteria for hangover headache. J Headache Pain (2004) 5:230-236 DOI 10.1007 Hangover headache: various manifestations and proposal for criteria. Vågå study of headache epidemiology Abstract Hangover headache has been focused upon in connection with the Vågå study of headache epidemiology, with 714 cases among 1122 dalesmen (64%). Most frequently the headache was global in location. It was more frequently located in the anterior (frontotemporal) than occipital area. A unilateral headache was present in only 3% of the dalesmen, many of whom were migraineurs. Headache seemed to be more intense in males than females. Headache usually seemed to be of a lower intensity than the pain of migraine, but higher than, or similar to, that of tension-type headache. The headache appeared late in the night/early morning hours, i.e., several hours after discontinuation of drinking. In most cases, headache stopped in the late morning hours, or at high noon, but it could even last until somewhat late in the afternoon; it rarely, if ever, exceeded 12 hours duration. Red wine seemed mostly to provoke headache the same evening (with traits similar to those of the spontaneously occurring headache attacks). Criteria for hangover headache are proposed.

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Section: Discussioncontrasting

confidence: 43%

Section: Discussionmentioning

confidence: 85%

Section: Discussionmentioning

confidence: 99%

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Hangover headache: various manifestations and proposal for criteria. Vågå study of headache epidemiology

Sjaastad

Bakketeig

2004

J Headache Pain

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“…A influência de fatores externos e internos desencadeando crises demonstra que mais de uma estrutura cerebral esta envolvida [10][11][12] . A influência positiva do NOM sobre esses mecanismos e sobre o número de crises foi comprovada nos estudos de Caputi 9 e descartada nos estudos de Bovim 7 .…”

Section: Discussionunclassified

Bloqueio anestésico do nervo occipital maior na profilaxia da migrânea

Piovesan

Werneck

Kowacs

et al. 2001

Arq. Neuro-Psiquiatr.

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RESUMO -Na fisiopatologia da enxaqueca muitas estruturas estão envolvidas, sendo que o nervo trigêmeo pode ser considerado a estrutura principal. Com o objetivo de determinar a influência do nervo occipital maior (NOM) sobre o comportamento da enxaqueca, estudamos 37 pacientes que apresentavam crises de enxaqueca. Utilizando-se de um estudo duplo cego cruzado os pacientes foram submetidos a infiltração do NOM com bupivacaína 0,5% (BP) e soro fisiológicos 0,9% (S.), os efeitos clínicos após os bloqueios anestésicos foram avaliados: subjetivamente através da escala visual analítica para dor e objetivamente determinou-se os limiares de percepção dolorosa. A comparação entre os dois grupos (BP-S.) e (S.-BP) mostrou que: o número e a duração das crises em todos os momentos do estudo não mudaram; a intensidade das crises no grupo (BP-S.) foi menor somente depois da segunda infiltração (P=0,020), em todos os outros momentos não se observaram alterações significativas. Concluímos que o bloqueio anestésico com BP sobre o NOM não altera o número e a duração das crises de migrânea, porém promove uma redução média na intensidade das crises 60 dias após a sua infiltração. Os resultados mostrados sugerem que o NOM participa ativamente sobre a modulação nociceptiva durante as crises de enxaqueca sem aura.PALAVRAS-CHAVE: bloqueio anestésico, nervo occipital maior, enxaqueca, profilaxia. Anesthetic blockade of the greater occipital nerve in migraine prophylaxisABSTRACT -Migraine comprises a great many encephalic structures in its pathophysiology with the trigeminal nerve (TN) type being one of the main ones. .or the purpose of determining a possible influence of the greater occipital nerve (GON) on migraine behavior, 37 patients who showed this pathology were studied. Using a double blind cross over group and submitting those patients to a GON infiltration with bupivacaina 0.5% (BP) and physiological serum 0.9% (PS), the clinical effects were evaluated: subjectively, through a pain analytical visual scale; objectively, by determining the threshold of pain perception (algometry). The comparison between the two groups (BP-PS) and (PS-BP) has shown that the number and duration of the attacks did not show significant statistical differences during the study. The intensity of the attacks was lower in group (BP-PS) only after the second infiltration (p=0.020), in the other moments no differences have been observed between the groups. The conclusion is that the anesthetic blockage with BP on the GON does not change the number of crises and their duration, but it does provokes an intensity reduction after 60 days from the infiltration. The results shown here suggest that GON participates in the cranial nociceptive modulation during crises of migraine without aura.KEY WORDS: blockade, greather occipital nerve, migraine, prophylaxis. O nervo occipital maior (NOM) é um dos responsáveis pela inervação sensorial cutânea da região occipital e suboccipital 1 . Pesquisas recentes mostraram que ele também participa ativamente na modulação nocice...

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“…Systematic surveys of patients attending the Princess Margaret Migraine Clinic at Charing Cross Hospital have established that about 18%-19% of patients report sensitivity to cheese or chocolate, and usually to both [1]. In a more recent epidemiological survey [2] the 18% of migrainous patients sensitive to all alcoholic drinks were distinguished from a further 12% sensitive only to red wine, but not to white wine or clear spirits.…”

Section: Introductionmentioning

confidence: 99%

Pharmacological analysis of red-wine-induced migrainous headaches

Peatfield¹,

Fletcher

Rhodes

et al. 2003

J Headache Pain

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IntroductionCheese, chocolate and red wine are the most publicised triggers for migrainous headaches, at least in English speaking countries. Systematic surveys of patients attending the Princess Margaret Migraine Clinic at Charing Cross Hospital have established that about 18%-19% of patients report sensitivity to cheese or chocolate, and usually to both [1]. In a more recent epidemiological survey [2] the 18% of migrainous patients sensitive to all alcoholic drinks were distinguished from a further 12% sensitive only to red wine, but not to white wine or clear spirits. There was a statistically significant but by no means absolute correlation between red wine and food sensitivity, though it was extremely rare for a diet-sensitive patient to be tolerant of all forms of alcohol [2]. We also found very few patients with tension or cluster headache to be sensitive to food or Abstract We describe a series of experiments designed to investigate the mechanisms by which headaches can be triggered by red wine in a small minority of migraine patients. Some red wines are particularly potent releasers of serotonin from platelet stores, but these are no more effective as triggers of headache in sensitive patients. Both the selective 5-HT 2A antagonist ketanserin and the non-selective 5-HT2ABC antagonist pizotifen blocked the majority of headaches, and we then thought the antihistamine properties of these two drugs might be important. In a third experiment, however, the H1 antagonist mepyramine did not convincingly antagonise the response to red wine. Plasma levels of the enzyme diamine oxidase, which metabolises histamine, were lower in all migraine patients, whether or not they were sensitive to red wine. The results reported here do not permit making firm conclusions; nevertheless it seems that different pharmacological receptors may be responsible in different patients.

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The Prevalence of Diet-Induced Migraine

Cited by 95 publications

References 11 publications

Hangover headache: various manifestations and proposal for criteria. Vågå study of headache epidemiology

Hangover headache: various manifestations and proposal for criteria. Vågå study of headache epidemiology

Bloqueio anestésico do nervo occipital maior na profilaxia da migrânea

Pharmacological analysis of red-wine-induced migrainous headaches

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