1993
DOI: 10.1001/archpsyc.1993.01820240031004
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The Prevalence of Seasonal Affective Disorder Is Low Among Descendants of Icelandic Emigrants in Canada

Abstract: This is the second study to find the prevalence of seasonal affective disorder and subsyndromal seasonal affective disorder to be lower among Icelanders or their descendants than among populations along the east coast of the United States. The results indicate that the relationship between prevalence of these disorders and geographic latitude is more complex than has previously been suggested; genetic adaptation in Icelandic populations may play an important role.

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Cited by 90 publications
(51 citation statements)
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“…Genetic type, gender and age are found to matter in SAD prevalence. For instance, Asians are more prone to summer-type SAD (Han et al, 2000;Ito et al, 1992) while for Caucasians it is more common to suffer from winter-type SAD (Magnússon & Axelsson, 1993;Soriano et al, 2007). Also, the highest propensity for SAD is found in women and younger individuals, compared with men and older individuals (Hedge & Woodson, 1996).…”
Section: Photoperiod Mood and Risk Aversionmentioning
confidence: 99%
“…Genetic type, gender and age are found to matter in SAD prevalence. For instance, Asians are more prone to summer-type SAD (Han et al, 2000;Ito et al, 1992) while for Caucasians it is more common to suffer from winter-type SAD (Magnússon & Axelsson, 1993;Soriano et al, 2007). Also, the highest propensity for SAD is found in women and younger individuals, compared with men and older individuals (Hedge & Woodson, 1996).…”
Section: Photoperiod Mood and Risk Aversionmentioning
confidence: 99%
“…These populations may, therefore, represent the best opportunity to assess the mental health of circumpolar peoples prior to the adoption of a more Western lifestyle, because careful scientific studies of mental health of past circumpolar populations is lacking (80,91). Low rates of SAD in circumpolar peoples may also have a genetic component (93).…”
Section: Circumpolar Peoplesmentioning
confidence: 99%
“…Epidemiologische Befunde sprechen dafür, dass einer SAD eine individuelle genetische Vulnerabilität zugrunde liegt [28].Außerdem konnte in Genetikstudien gezeigt werden, dass SAD-Patienten an der Steuerungsregion des Serotonintransporters (5-HTTLPR) häufi-ger das kurze Allel aufweisen [34]. Da aber möglicherweise ein exogener Lichtmangel die Manifestation einer SAD triggert [9,10,43], wurden schon früh Untersuchungen über die Prävalenz und klinische Ausprägung in unterschiedlichen geographischen Regionen und Populationen durchgeführt [4,7,11,20,29,30,31,32,45,53].…”
Section: Schlüsselwörterunclassified