The tako–tsubo syndrome and bronchial asthma: the chicken or the egg dilemma

Pontillo, Daniele; Patruno, Nicolino; Stefanoni, R

doi:10.2459/jcm.0b013e32833cdbb0

Cited by 10 publications

(6 citation statements)

References 2 publications

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“…Diabetes mellitus leads to neuroautonomic nerve remodeling and an up-regulation of vasoactive neuropeptides like NPY, which may lead to enhanced susceptibility to stress cardiomyopathy and arrhythmias (46)(47)(48). Asthma exacerbation is another possible trigger of stress cardiomyopathy, mainly following medical interventions (short acting ß2 adrenergic receptor agonist, epinephrine and intubation) (49,50). The etiology of stress cardiomyopathy in the course of an asthma attack is not understood.…”

Section: Risk Factorsmentioning

confidence: 99%

Stress Cardiomyopathy Diagnosis and Treatment

Chazal

Buono

Keyser-Marcus

et al. 2018

Journal of the American College of Cardiology

429

410

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Section: Risk Factorsmentioning

confidence: 99%

Stress Cardiomyopathy Diagnosis and Treatment

Chazal

Buono

Keyser-Marcus

et al. 2018

Journal of the American College of Cardiology

429

410

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“…Catecholamines (e.g., noradrenaline) and stress‐related neuropeptide Y are believed to play central roles in TS causing not only cardio‐inhibitory effects but also acute microvascular dysfunction 3 . Interestingly, asthma exacerbation, especially after treatment (short‐acting ß2 adrenergic receptor agonist, adrenaline and intubation), is well described as a TS trigger 4, 5 …”

Section: Discussionmentioning

confidence: 99%

“…3 Interestingly, asthma exacerbation, especially after treatment (shortacting ß2 adrenergic receptor agonist, adrenaline and intubation), is well described as a TS trigger. 4,5 Kounis syndrome (KS) is an acute coronary syndrome precipitated by severe allergic events. In KS, it is postulated that mast cell mediators such as histamine, platelet-activating factor, cytokines, and so on cause coronary artery spasm, atheromatous plaque rupture or coronary artery stent thrombosis.…”

Section: Discussionmentioning

confidence: 99%

Transfusion double whammy? Adrenaline‐takotsubo‐anaphylaxis‐Kounis complex post transfusion?

Badami¹

2022

Vox Sanguinis

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Background and Objectives: The adrenaline-takotsubo-anaphylaxis-Kounis, or the ATAK complex, where there are clinical and pathophysiological overlaps between takotsubo and Kounis syndromes, in which histaminergic, adrenergic and other mediators may play roles, was recently described. The objective of this report was to describe three cases where the ATAK complex was suspected to have occurred after transfusion.Materials and Methods: Three cases were recently reported to the New Zealand Blood Service haemovigilance programme that appeared to have features in common suggestive of the ATAK complex.Results: All three patients had had a blood component transfused, an initial severe allergic reaction, treatment with adrenaline or a congener, subsequent acute left ventricular failure or transfusion-associated circulatory overload, and features suggestive of takotsubo cardiomyopathy.Conclusions: Although rarely described, transfusion-associated ATAK complex may be occurring more often than believed. Circumstances during a transfusion may predispose to it. It should be suspected if the sequence of events described above occur. Its characteristics need to be better understood. Risk factors for it may be modifiable.

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“…One of the non-cardiac risk factors frequently associated with stress CM is asthma, particularly after the use of medical interventions such as beta-2 agonists, racemic epinephrine, and intubation. It is not fully understood whether it is the asthma itself or the treatment that triggers the syndrome (18). Marijuana use is also associated with stress CM with a subsequent higher mortality (19).…”

Section: Risk Factorsmentioning

confidence: 99%

Cardiovascular Imaging in Stress Cardiomyopathy (Takotsubo Syndrome)

Zghyer

Botheju

Kiss

et al. 2022

Front. Cardiovasc. Med.

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Stress cardiomyopathy (Takotsubo syndrome) is a reversible syndrome stemming from myocardial injury leading to systolic dysfunction and is usually noted in the setting of a stressful event, be it an emotional or physical trigger. While the exact pathophysiology behind stress cardiomyopathy is yet unknown, there is ample evidence suggesting that neurocardiogenic mechanisms may play an important role. Although historically stress cardiomyopathy was generally thought to be a relatively benign condition, there is growing recognition of the cardiovascular complications associated with it despite its reversibility. Our review aims to shed light onto key cardiovascular imaging modalities used to diagnose stress cardiomyopathy while highlighting the role that imaging plays in assessing disease severity, identifying complications, dictating treatment approaches, and in short-term and long-term prognosis.

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The tako–tsubo syndrome and bronchial asthma: the chicken or the egg dilemma

Cited by 10 publications

References 2 publications

Stress Cardiomyopathy Diagnosis and Treatment

Stress Cardiomyopathy Diagnosis and Treatment

Transfusion double whammy? Adrenaline‐takotsubo‐anaphylaxis‐Kounis complex post transfusion?

Cardiovascular Imaging in Stress Cardiomyopathy (Takotsubo Syndrome)

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