Thyrotoxic Hypercalcemia Treated with Corticosteroids

Aanderud, Sylvi; Bassøc, Hans H.

doi:10.1111/j.0954-6820.1980.tb09761.x

Cited by 3 publications

(1 citation statement)

References 18 publications

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“…In the remaining cases, it was impossible to determine which came first. 22 However, this is complicated with the fact that the symptoms of thyrotoxicosis such as loose stools or diarrhea and increased appetite may mask the symptoms of hypercalcemia such as constipation, nausea, and anorexia. 14 In managing patients with biochemical changes of TT and PHPT, one author recommended that a symptomatic hypercalcemia lead to further examination for PHPT.…”

Section: Causal Relationsmentioning

confidence: 99%

Type 2 Diabetes Mellitus, Primary Hyperparathyroidism, and Thyrotoxicosis Diagnosed Simultaneously in 2 Patients

Hansen¹,

Vestergaard²

2007

The Endocrinologist

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Type 2 diabetes mellitus (T2D) and thyroid diseases (TD) are common endocrine diseases. They often occur in the same patient concomitantly by chance. However, they occur together more often than expected from the incidences in the general population. Primary hyperparathyroidism (PHPT) is not as common, but it is seen more often in patients with T2D or TD than in the general population as a whole. There are indications that the 3 diseases might have a connection or a causal relation of some kind. We present 2 cases in whom the 3 diseases were diagnosed together.

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Section: Causal Relationsmentioning

confidence: 99%

Type 2 Diabetes Mellitus, Primary Hyperparathyroidism, and Thyrotoxicosis Diagnosed Simultaneously in 2 Patients

Hansen¹,

Vestergaard²

2007

The Endocrinologist

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Mineral Metabolism in Health and Disease

Agus

Attie

Goldfarb

et al. 1983

Contemporary Nephrology

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Coexisting hyperparathyroidism with thyrotoxicosis

McGowan

Vaswani

Shperling

1991

J Endocrinol Invest

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The coexistence of hyperparathyroidism complicating thyrotoxicosis is quite rare. We report the case of one patient who presented with thyrotoxicosis, (total thyroxine of 15.1 micrograms/dl (5-13), free thyroxine index of 18 (4-15) and triiodothyronine by RIA of 305 ng/dl (70-230) and asymptomatic hypercalcemia of 15 mg/dl (8.5-10.6), who was also initially noted to have an elevated (C-terminal) serum immunoreactive parathyroid hormone (iPTH) level of 8,800 pg/ml (50-340). With propylthiouracil and propranolol, however, this patient became normocalcemic with a decrease in iPTH values to 714 pg/ml. As the patient was tapered from medication, after being rendered euthyroid, a recurrence of hypercalcemia with rising iPTH levels occurred. PTH levels should be helpful in defining coexisting hyperparathyroidism in patients with thyrotoxicosis since in the latter iPTH is usually suppressed. Our findings support the recommendation that in patients suspected of having both hyperparathyroidism and hyperthyroidism, a diagnosis of the former can only be made with certainty after the patient has been rendered euthyroid with persistently elevated serum calcium and iPTH levels. While there are no clinical features which permit the easy identification of patients who present with dual lesions, the determination of iPTH values may be the most consistently helpful test initially, whereas other parameters such as vitamin D, serum phosphate are less reliable.

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Thyrotoxic Hypercalcemia Treated with Corticosteroids

Cited by 3 publications

References 18 publications

Type 2 Diabetes Mellitus, Primary Hyperparathyroidism, and Thyrotoxicosis Diagnosed Simultaneously in 2 Patients

Type 2 Diabetes Mellitus, Primary Hyperparathyroidism, and Thyrotoxicosis Diagnosed Simultaneously in 2 Patients

Mineral Metabolism in Health and Disease

Coexisting hyperparathyroidism with thyrotoxicosis

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