toxoid, human tetanus immunoglobulin, and penicillin G intravenously as the usual treatment. Although he was conscious and alert, he exhibited signs of lockjaw, which progressed to opisthotonos 10 h after admission. The patient's muscle spasms became so severe that it was necessary to intubate his trachea, using intravenous midazolam and vecuronium bromide to allow controlled ventilation.After the onset of severe muscle spasms, cardiovascular instability occurred frequently. Despite the administration of intravenous diltiazem hydrochloride 5 mg hourly and nicardipine 1 mg as required, the patient's hemodynamic state remained unstable, and his systolic blood pressure exceeded 200 mmHg on several occasions. His condition was classified as very severe tetanus, as graded by modified Ablett's classification [1,2].One week following admission, the patient suddenly developed hypotension and tachycardia (Fig. 1). Although he had often shown the alternate hypertension and hypotension caused by sympathetic storm, we suspected hemorrhagic hypotension in this situation. This suspicion was confirmed by a decrease in hemoglobin concentration from 12.5 to 10.7 g·dl Ϫ1 after fluid resuscitation. A massive intraperitoneal hematoma was detected by abdominal ultrasonography and computed tomography. The emergency arteriography showed bleeding from multiple SMA aneurysms (Fig. 2). We used a 5-Fr catheter (Mallinckrodt) via the right femoral artery. Embolization was then performed with eight coils Cook), gelatine (Gelfoam), and 3-0 silk strings. After embolization for the aneurysms of the main trunk, the patient's hemodynamic state returned to a normal range (Fig. 2).Temporary paralytic ileus resulted from the embolization and the peritoneal hematoma, requiring treatment with sodium picosulfate orally, panthenol and dinoprost (PGF 2α ) intravenously, and glycerin