Tumour growth and nitrogen metabolism in the host (Review).

Argilés, Josep Μ.; Costelli, Paola; Carbó, Neus; Pallarés-Trujillo, J.; López‐Soriano, Francisco J.

doi:10.3892/ijo.14.3.479

Cited by 11 publications

(9 citation statements)

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“…Most of this wasting occurs in skeletal muscle, appears to be due to accelerated muscle protein degradation, and is unresponsive to supplementary feeding [69]. This wasting is attributed in large part to the actions of a number of cytokines including TNF-α, IL-1, IL-6, and IFN-γ [3].…”

Section: Muscle Mass and Inflammationmentioning

confidence: 98%

Possible links between behavioral and physiological indices of tiredness, fatigue, and exhaustion in advanced cancer

Olson

Turner

Courneya

et al. 2007

Support Care Cancer

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Section: Muscle Mass and Inflammationmentioning

confidence: 98%

Possible links between behavioral and physiological indices of tiredness, fatigue, and exhaustion in advanced cancer

Olson

Turner

Courneya

et al. 2007

Support Care Cancer

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“…Muscle protein hypercatabolism in cancer cachexia is paralleled by modulations in the expression of components of the ATP-ubiquitin-dependent proteolytic pathway (reviewed in [33]). Recent data suggest that the activity of this degradative system is indeed enhanced in cancer cachexia, proteasome chymotrypsin-like activity being increased in the skeletal muscle of rats bearing the Yoshida AH-130 hepatoma [11 . ].…”

Section: Muscle Wasting In Sepsis and Cancer Cachexiamentioning

confidence: 99%

Mechanisms of skeletal muscle depletion in wasting syndromes: role of ATP-ubiquitin-dependent proteolysis

Costelli

Baccino

2003

Current Opinion in Clinical Nutrition &Amp; Metabolic Care

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As the present overview points out, several questions still remain unanswered in the issue of muscle wasting. While many different signals that have the potential to enforce the acceleration of muscle protein breakdown have been identified, it is largely unknown how they are transduced and converge into the hypercatabolic response and how the proteolytic pathways involved are activated. The concept seems to emerge that there may be a coordinated action of different proteolytic pathways in setting up muscle protein turnover towards excess catabolism.

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“…Cytokines; TNF-α, IL-1, IL-6 and IFN-γ have been implicated in the induction of cancer related muscle wasting. 34 …”

Section: The Role Of Cytokinesmentioning

confidence: 99%

Cancer cachexia-anorexia syndrome and skeletal muscle wasting

Jurdana

2009

Radiology and Oncology

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Tumour growth and nitrogen metabolism in the host (Review).

Cited by 11 publications

References 0 publications

Possible links between behavioral and physiological indices of tiredness, fatigue, and exhaustion in advanced cancer

Possible links between behavioral and physiological indices of tiredness, fatigue, and exhaustion in advanced cancer

Mechanisms of skeletal muscle depletion in wasting syndromes: role of ATP-ubiquitin-dependent proteolysis

Cancer cachexia-anorexia syndrome and skeletal muscle wasting

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