Computational and learning theory models propose that behavioral control reflects value that is both cached (computed and stored during previous experience) and inferred (estimated on-the-fly based on knowledge of the causal structure of the environment). The latter is thought to depend on the orbitofrontal cortex. Yet, some accounts propose that the orbitofrontal cortex contributes to behavior by signaling “economic” value, regardless of the associative basis of the information. We found that the orbitofrontal cortex is critical for both value-based behavior and learning when value must be inferred but not when a cached value is sufficient. The orbitofrontal cortex is thus fundamental for accessing model-based representations of the environment to compute value rather than for signaling value per se.

The fundamental function of information technology (IT), whether internal or external, is the costeffective provision of IT services that meet organizational needs and align with organizational strategy. With ever-increasing adoption of IT management "best practices," industry now leads the academic community by recognizing the need for IT professionals educated in the IT service management (ITSM) processes-processes directly associated with the delivery of high-quality, cost-effective IT services. This article presents a summary of panel member presentations and subsequent discussions at Americas Conference on Information Systems (AMCIS) 2007 concerning the need to, and means for, incorporating ITSM concepts into business-school MIS/IS/CIS curricula. Panel members from industry strongly advocated the importance of including ITSM principles and concepts within business-school IS curricula. Academic members of the panel also articulated this need and described initiatives at their and other schools where ITSM concepts have been or are planned for inclusion in business school curricula at both the graduate and undergraduate levels. ITSM research opportunities, faculty preparedness, and possible resistance from within the IS academic community were additionally addressed.

We propose QuadMixer, a novel interactive technique to compose quad mesh components preserving the majority of the original layouts. Quad Layout is a crucial property for many applications since it conveys important information that would otherwise be destroyed by techniques that aim only at preserving shape. Our technique keeps untouched all the quads in the patches which are not involved in the blending. We first perform robust boolean operations on the corresponding triangle meshes. Then we use this result to identify and build new surface patches for small regions neighboring the intersection curves. These blending patches are carefully quadrangulated respecting boundary constraints and stitched back to the untouched parts of the original models. The resulting mesh preserves the designed edge flow that, by construction, is captured and incorporated to the new quads as much as possible. We present our technique in an interactive tool to show its usability and robustness.

It has been proposed that schizophrenia results, in part, from the inappropriate or spurious attribution of salience to cues in the environment. We have recently reported neural correlates of salience in the basolateral amygdala (ABL) of rats during learning in an odor-guided discrimination task. Here we tested whether this dopamine-dependent salience signal is altered in rats with neonatal ventral hippocampal lesions (NVHLs), a rodent model of schizophrenia. We found that ABL signals related to violations in reward prediction were only mildly affected by NVHL; however, neurons in rats with NVHLs showed significantly stronger selectivity during odor sampling, particularly for the more salient large-reward cue. The elevated cue-evoked activity in NVHL rats was correlated with heightened orienting behavior and also with changes in firing to the shifts in reward, suggesting that it reflected abnormal signaling of the large rewardpredicting cue's salience. These results are broadly consistent with the proposal that schizophrenics suffer from enhanced signaling of salience.

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