Tea is an important beverage consumed worldwide. It is a source of important secondary metabolites like catechins monoterpenoids, carotenoids etc. Catechins are responsible for the beneficial health effects of tea. Over the decades conventional tea cultivation practice using synthetic fertilizer and pesticides has jeopardize the soil health particularly due to micronutrient deficiency, instability in yield and reduced product quality. Such threat has led to emergence of organic farming practice for improvement of crop yield and product quality that finally has an impact on human health defence. Field experiments conducted using organic farming practice has shown improved soil health by improving availability of micronutrients which in terms improved crop yield and quality of tea. The content of secondary phenolics compounds like total phenolics, GCG, EGCC, ECG was higher in when tea grown organically. Antioxidant property of tea extract from different farming practice was studied by DPPH method resulting higher radical scavenging capacity than tea grown conventionally. From the pharmacological study the organic tea extract has shown better control of Parkinsonism in two different animal model experiments. Organic tea extract exhibited better performance in reduction of Superoxide dismutase and catalase activity in brain in Parkinson's disease induced in test mice model than the tea extract obtained from conventional farming practice. The value of SOD and catalase activity in MPTP induced mice model given tea extract grown in control, conventional farming, vermicompost and vermicompost+vermiwash treatment are respectively 1.31 ± 0.16 c , 1.8 ± 0.16 d , 0.95 ± 0.14 e , 0.79 ± 0.06; and 0.98 ± 0.07 c , 1.10 ± 0.07 d , 0.78 ± 0.07 e , 0.69 ± 0.05 a for Tv 25 variety. Similar result is obtained for Tv1 variety also.The present study generated information on soil, crop performance, yield and quality of tea related to health defense, which should be regarded as valuable information for a perennial crop like tea.

Tea (Camellia sinensis) having anti-in ammatory, antioxidant, and free radical scavenging properties, may be bene cial to prevent the symptoms of neurodegenerative disorders like Alzheimer's disease (AD). In this present study, eld experiments using the productive tea clone (TV25) with four nutrient management treatments were conducted during 2015 to 2017 in the research farm of Agricultural and

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Tea (Camellia sinensis) having anti-inflammatory, antioxidant, and free radical scavenging properties, may be beneficial to prevent the symptoms of neurodegenerative disorders like Alzheimer’s disease (AD). In this present study, field experiments using the productive tea clone (TV25) with four nutrient management treatments were conducted during 2015 to 2017 in the research farm of Agricultural and Food Engineering Department, Indian Institute of Technology Kharagpur. The four nutrient management treatments were no application of fertilizer (control), organic fertilizer (OF), inorganic fertilizer (IF), and integration of OF and IF (IF+OF). The total phenolic content of tea leaves in terms of gallic acid equivalent (GAE) in control, OF, IF, IF+OF were 263±2.4 mg/g, 292±1.6mg/g, 203±2.43 mg/g ,288.6±2.11 mg/g, respectively. Tea leaf samples of these treatments were fed to the intracerebroventricular (ICV) colchicine administered rats. The animal study was double-blinded and randomized. Assessment of anxiety status was done for the rat model in an elevated open field with a novel object in two intervals (14-day and 21-day study). Anxiolytic behaviour with the lower corticosterone (CORT) level (82ng/ml) was observed in ICV colchicine administered rat models of AD. After feeding of organically and inorganically grown tea extract (20 mg/kg) for 14 days and 21days, it was found that the anxiolytic behaviour decreased with the increased concentration of serum CORT. However, organic tea showed greater increase in CORT level (216.1 ng/ml) as compared to inorganic tea (214 ng/ml). Thus, this study showed organic tea may act as a favourable agent or adjuvant in the improvement of the anxiolytic behaviour in rat model of AD.

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