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Increased left ventricular mass (LVM), LVM index (LVMI) and left ventricular hypertrophy (LVH) are significantly associated with a higher incidence of clinical events including death. 1 Also, although increased LVMI and LVH may be associated with traditional cardiovascular risk factors, the presence of LVH predicts adverse cardiovascular events independent of underlying cardiovascular disease and risk factors. 1,2 However, the association of coronary anatomical parameters with LVM and LVMI remains unclear.Coronary artery dominance defines the anatomical variations in the coronary circulation. While right dominance (RD) is the most prevalent coronary circulation pattern in the general population and is seen in approximately 70%-80% of the population, co-dominance (CD) and left dominance (LD) patterns are seen in approximately 10%-20% and 10% of the population, respectively. 3 Although

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Introduction: Transradial coronary angiography (TRA) is associated with a lower incidence of bleeding rate and access site complications and is associated with better outcomes compared to transfemoral angiography. However, radial artery spasm (RAS) is an important limitation of TRA procedures. Little is known regarding the relationship of serum vasodilator and inflammatory markers with RAS. Therefore, the present study aimed to investigate the association between serum adropin level and RAS in patients undergoing TRA. Methods: From February 2020 to January 2021, 39 consecutive patients who underwent elective daiagnostic TRA and experienced RAS during the procedure, and 42 age and sex matched controls who did not experience RAS were prospectively included into the study. The groups were compared regarding serum adropin levels and inflammatory markers. Results: Although adropin levels were found to be lower in the RAS group, this difference was not statistically significant between the the patients with RAS and controls (14.9 vs. 16.1, P=0.105). However, inflammatory parameters monocyte count and MHR (monocyte/HDL cholesterol ratio) were found to be statistically significantly higher in the RAS group compared to controls (P=0.001 and P=0.010, respectively). Moreover, a significant positive correlation was found between the monocyte count and RAS (r:0.360, P<0.001), and between MHR and RAS (r:0.288, P=0.009). Furthermore, multivariate analysis demonstrated that monocyte count (OR:1.671, 95%CI:1.312-2.094, P=0.001) and MHR (OR:1.116, 95%CI:1.054-1.448, P=0.022) were found to be independent predictors of RAS. Conclusion: Serum vasodilator and inflammatory markers may be useful in the prediction of RAS in patients undergoing TRA procedures.