The human stress response has been characterized, both physiologically and behaviorally, as "fight-or-flight." Although fight-or-flight may characterize the primary physiological responses to stress for both males and females, we propose that, behaviorally, females' responses are more marked by a pattern of "tend-and-befriend." Tending involves nurturant activities designed to protect the self and offspring that promote safety and reduce distress; befriending is the creation and maintenance of social networks that may aid in this process. The biobehavioral mechanism that underlies the tend-and-befriend pattern appears to draw on the attachment-caregiving system, and neuroendocrine evidence from animal and human studies suggests that oxytocin, in conjunction with female reproductive hormones and endogenous opioid peptide mechanisms, may be at its core. This previously unexplored stress regulatory system has manifold implications for the study of stress.
Important features of the self-concept can be located outside of the individual and inside close or related others. The authors use this insight to reinterpret data previously said to support the empathyaltruism model of helping, which asserts that empathic concern for another results in selflessness and true altruism. That is, they argue that the conditions that lead to empathic concern also lead to a greater sense of self-other overlap, raising the possibility that helping under these conditions is not selfless but is also directed toward the self. In 3 studies, the impact of empathic concern on willingness to help was eliminated when oneness-a measure of perceived self-other overlapwas considered. Path analyses revealed further that empathic concern increased helping only through its relation to perceived oneness, thereby throwing the empathy-altruism model into question. The authors suggest that empathic concern affects helping primarily as an emotional signal of oneness.
When pressure to perform is increased, individuals commonly perform worse than if there were no pressure ("choking under pressure'). Two mechanisms have been proposed to account for this effect-distraction (cognitive load), wherein pressure distracts attention from the task, and self focus, wherein attention shifts inward interfering with performance. To distinguish between these two competing explanations, the current experiment manipulated pressure by offering performance-contingent rewards. For half the participants, cognitive load was increased by requiring participants to count backward from 100. Additionally, adaptation to self awareness was manipulated by videotaping half the participants during practice trials. Results show that pressure caused choking when participants were not distracted and had not been adapted to self awareness. This effect was attenuated when cognitive load was increased or when self-awareness adaptation had occurred. These results support self focus mediated misregulation as the mechanism for choking and disconfirm the distraction hypothesis.
Important features of the self-concept can be located outside of the individual and inside close or related others. The authors use this insight to reinterpret data previously said to support the empathy-altruism model of helping, which asserts that empathic concern for another results in selflessness and true altruism. That is, they argue that the conditions that lead to empathic concern also lead to a greater sense of self-other overlap, raising the possibility that helping under these conditions is not selfless but is also directed toward the self. In 3 studies, the impact of empathic concern on willingness to help was eliminated when oneness--a measure of perceived self-other overlap--was considered. Path analyses revealed further that empathic concern increased helping only through its relation to perceived oneness, thereby throwing the empathy-altruism model into question. The authors suggest that empathic concern affects helping primarily as an emotional signal of oneness.
This study was designed to investigate whether post-exercise analgesia occurs following an ad lib exercise routine. All of the 17 male participants exercised on a regular basis. In an exercise setting (student gymnasium) they participated in 20 min of self-selected exercise, while in the neutral setting (laboratory) they rested quietly for 20 min. Pain was induced via the gross pressure device. Pain threshold and pain tolerance were measured twice, with an interval of 20 min, in both the exercise and the neutral setting. Pain threshold was stable in the exercise setting. A significant increase in pain tolerance followed the 20 min bout of exercise, indicating a post-exercise analgesic response. These results support the prediction that the analgesic effect of exercise is not limited to controlled experimental conditions, but generalizes to naturally occurring situations.
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