No abstract

Oral extramedullary plasmacytomas (EMP) are locally aggressive tumors in the dog that rarely metastasize. They represent 5.2% of all canine oral tumors and 22.0 to 28.0% of all EMPs diagnosed are in the oral cavity. EMPs consist of neoplastic plasma cells that do not arise from the bone marrow. No relationship between EMP and the development of multiple myeloma has been determined in dogs. Complete surgical excision is the primary treatment for this neoplasm and is usually curative. Multiple oral EMPs within the same patient have been rarely reported with tumors arising in the same location in the mouth. To the authors' knowledge, multicentric oral EMP, as described in the following cases, has not been reported in the dog.

Of the six recognized types of tooth luxation injuries, intrusion generally carries the most guarded long-term prognosis due to the high risk of complications, including root resorption, ankylosis, marginal bone loss, and pulp canal obliteration or necrosis. The degree of traumatic intrusion and stage of root development affects the outcome and treatment planning. This report describes the clinical history, oral and radiographic examination findings, and rationale for surgical extraction in 2 dogs and 1 cat with traumatic intrusion of a maxillary canine tooth.

An oral topically applied gel with essential oils and polyphenolic antioxidants applied daily after an initial professional dental cleaning decreased oral malodor in dogs.

A two-year-old Boerboel dog presented for a discolored left maxillary canine tooth. Dental radiographs revealed abnormally mineralized pulp within the discolored tooth. Similar radiographic findings were also seen in both maxillary third incisor teeth and in the remaining canine teeth to varying degrees. The discolored tooth was treated by surgical extraction and histopathology revealed abnormal dentin deposition within the canal indicative of dentin dysplasia. Although not previously documented in the dog, the pulpal changes in multiple teeth of the dog reported here were similar to those described for odontoblastic dysplasia in humans. This case report includes a review of developmental abnormalities of dentin in humans and pulpal response to inflammation and injury.