Objective: Taurodontism is a dental anomaly characterized by an enlarged pulp chamber and apycal displacement of the pulpar floor. The prevalence of taurodontism in normal population is controversial. It has been reported that taurodontism is frequently observed in Klinefelter’s patients. The purpose of this study was to assess the prevalence of taurodontism in a group of Italian Klinefelter’s patients and in a randomly selected male population of Italy and to compare the results with published data. Materials and methods: Digital panoramic radiographs of 16 Klinefelter’s patients and of 100 normal males were retrospectively studied in order to investigate the prevalence of taurodontism in these groups of patients. Results: Taurodont teeth were observed in 2 of the 16 Klinefelter’s patients (12.5%) and in 2 of 100 normal males (2.0%). Conclusions: Our results confirm the higher prevalence of taurodontism in Klinefelter’s patients compared to the normal population (12.5% vs. 2.0%). Due to the wide discrepancy of incidence of taurodontism reported in literature (0.04%-48.0% in normal population; 12.5%-88.0% in Klinefelter’s patients), we conclude that it is not possible to state which is the prevalence of taurodontism in a normal population nor among Klinefelter’s patients.

Axillary lymphadenopathy is defined as abnormality (e.g., increase in size or density) of lymph nodes in the armpit, caused by malignant diseases such as metastases (mainly from primary breast cancer), lymphoma, or leukaemia as well as benign conditions such as infectious or autoimmune systemic diseases. Appropriate imaging and pathological examinations on needle samples, together with accurate clinical correlation are needed for a correct diagnosis and management. Herein, we report a case of a 47-year-old female presented at our department of radiology for her annual mammographic screening. Mammography demonstrated multiple bilateral, enlarged, even although benign-appearing axillary lymph nodes. While both breasts showed no sign of malignancy on mammograms, the lymphadenopathies suggested a potential underlying inflammatory process. Previous mammography performed five years before did not present any lymphadenopathy. The patient, recalled for additional breast and axillary ultrasound and for clinical correlation, reported that she had been suffering for at least four years from an autoimmune systemic disease, mixed connective tissue disease, recently overlapping with psoriatic arthropathy, thus explaining the aetiology of reactive lymph nodes enlargement.

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Background Our purpose was to evaluate the correlations between right ventricular (RV) late gadolinium enhancement (LGE) at cardiac magnetic resonance (CMR) in patients with tetralogy of Fallot (ToF) scheduled for pulmonary valve replacement (PVR) and post-PVR functional data. Methods We retrospectively reviewed ToF patients scheduled for PVR who underwent two CMR examinations at our institution, one before the procedure (CMR-0), including contrast-enhanced sequences, and one after the procedure (CMR-1). Functional left and RV data were obtained by segmenting short-axis stacks on both CMR examinations, and normalised variations were calculated by dividing differences between CMR-1 and CMR-0 by the intercurring time interval, whereas the RV scar burden was assessed on CMR-0 LGE sequences both semiquantitatively and quantitatively. Data were reported as median and interquartile range, differences were appraised with the Mann–Whitney U test, while correlations were assessed with Spearman’s ρ. Results Fifteen patients with a median age of 25 years (16–29), including 9 (60%) males, with a median time interval between CMR-0 and CMR-1 of 17 months (12–23), were retrospectively reviewed. The semiquantitative LGE score at CMR-0 was 7 (6–9), and LGE volume was 4.49 mL (3.70–5.78), covering 5.63% (4.92–7.00) of the RV. RV LGE score showed a moderate positive correlation with the normalised variation of RV stroke volume (ρ = 0.662, p = 0.007) and a borderline moderate positive correlation with the normalised variation of RV end-diastolic indexed volume (ρ = 0.513, p = 0.050). Conclusions The assessment of RV LGE before PVR may provide insights on post-PVR functional data, potentially facilitating a patient-tailored treatment pathway.

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