Ectopic pregnancy accounts for approximately 2% of all pregnancies and is the most common cause of pregnancy-related mortality in the first trimester. Initial evaluation consists of β-hCG and pelvic ultrasonography. The fallopian tube is the most common location for an ectopic pregnancy. Other types of ectopic pregnancy include cornual, ovarian, cervical, scar, and abdominal pregnancy. In very rare cases, the abdominal pregnancy may be retroperitoneal. The diagnosis is seldom established before surgery and therapy is surgical resection of the ectopic mass. A 26-year-old woman visited our emergency department with sudden massive vaginal bleeding. She had undergone curettage 3 weeks before. But the transvaginal sonogram of the cul-de-sac revealed no fl uid collection. She failed variable-dose methotrexate therapy. On laparoscopic operation, we found a cystic, conception-like structure in the retroperitoneum. Histology of resected structure showed chorionic villi. We describe this case with a brief review of the literature.

Sacrococcygeal teratoma (SCT) is a rare subset of germ cell neoplasm and occurs in approximately 1 in 35,000 live births. Most SCTs are benign, but about 20% are malignant. They originate from totipotent cells from Hansen's node or primitive germ cells, but the exact etiology remains uncertain. Antenatal diagnosis of SCT can be made by ultrasound. The fetus with SCT remains at high risk for perinatal complications and death. Perinatal mortality and morbidity are most strongly related to high-output cardiac failure because of arteriovenous shunting within the tumor, subsequent fetal hydrops, polyhydramnios, and preterm delivery. Recently we have experienced a case of immature SCT with hydrops and polyhydrmnios diagnosed by prenatal ultrasonography at 21+5 weeks, resulted in stillbirth. We describe this case with a brief review of the literature.

Lymphogranuloma venereum (LGV) is a rare sexually transmitted disease caused by chlamydia trachomatis serotypes L1-L3. The peak incidence occurs in persons 29 to 47 years old, and men are six times more likely than women to manifest clinical infection. The disease clinically manifests in three stages: a primary lesion consisting of a superfi cial ulcer or erosion, a secondary stage with prominent lymphadenopathy and a later stage with disease manifestations such as fi brosis, fi stula, and anorectal strictures. The diagnosis of LGV is based on clinical suspicion, epidemiologic information, and the exclusion of other causes of inguinal lymphadenopathy. Diagnosis of LGV may be difficult. Patients with a clinical syndrome consistent with LGV, including genital ulcer with lymphadenopathy, should be treated for LGV. Recently we have experienced a case of LGV in 44-years-old women with inguinal bubo and sclerosing fi brous skin. We treated her with doxycycline and advancement fl ap.