Purpose: To estimate statewide presentation delay, misdiagnosis rate, inter-hospital transfer times and testicular salvage for testicular torsion patients treated in our state's public health system. Patients and Methods: Case series of consecutive testicular torsion patients treated in our state's public health system between 2012-2018. Predictors included presentation delay (time from symptoms to first medical assessment), facilitie's level-of-care (primary, secondary, tertiary), first diagnosis (torsion, epididymitis, other), Doppler-enhanced ultrasound request (Doppler-US) and inter-hospital transfer times, with surgical organ salvage as the main response. We used Bayesian regression to estimate the effect of first examining facilitie's level-of-care, first diagnosis, and Doppler-US on transfer time. Results: 505 patients were included, most (298, 59%) with presentation delay >6 hours. Misdiagnosis at first examining facility raised transfer time from median 2.8 to 23.4 (epididymitis) and 37.9 hours (other) and lowered testicular salvage rates from 60.3% (torsion) to 10.7% (epididymitis) and 18.3% (other). Doppler-US had negligible effects on transfer time once controlling for misdiagnosis in the regression model. Although organ salvage in patients presenting before 6 hours at the tertiary facility was high (94.6%, and about 20% lower for those presenting at lower levels-of-care), the overall salvage rate was more modest (46%). Conclusion: Our low overall testicular salvage rates originated from a large proportion of late presentations combined with long transfer times caused by frequent misdiagnoses. Our results indicate that efforts to improve salvage rates should aim at enhancing populationwide disease awareness and continuously updating physicians working at primary and secondary levels-of-care about scrotal emergencies.
The article describes the authors' 20-year experience with penile fracture, gained from their work at largest emergency department of the city of Rio de Janeiro (1). This study follows previous publications on the subject of penile fracture by the same group of authors (2-4), and provides the practising urologist with valuable information about the diagnosis and treatment of the disease. Although there is justifiable debate about whether one should surgically approach penile fractures through a subcoronal (the authors'-and ours'-choice), penoescrotal or perineal incision, the attending urologist must first determine whether the patient has, in fact, a penile fracture, or a vascular injury mimicking a penile fracture, i.e. a false penile fracture (5-7). The authors' no-nonsense approach to this diagnostic problem was to consider patients that did not describe immediate detumescence after injury, presenting small-to-moderate penile hematoma (or none at all) and without abnormalities in the palpation of the corpora cavernosa as having a lower likelihood of penile fracture-and offer to these patients supplemental imaging studies or conservative treatment with close follow-up. The reader must be cautioned, however, that such line of action is grounded by the authors' large experience, and that the default approach in clinically diagnosed penile fracture patients is surgical exploration. Another facet of the authors' large experience with the disease can be observed in their approach to synchronous urethral injuries, diagnosed in 54 of their patients (18.7%). Preoperative retrograde urethrography (RUG) was performed in a little less than half of those (25/54, 47%), and their logic in restricting the indication of RUG is simple to follow: Urethral injuries are not rare in penile fracture; RUG has been associated with high false-negative rates (reference 14 in the article); penile degloving provides excellent exposure to the penile urethra; urethral repair can be easily performed concomitant with tunical repair; ergo, one can forgo RUG in these cases. Yet, one should not promptly waive RUG if the surgical plan involves an incision directly to the site of injury, even more so if there is any sign of urethral injury, e.g. urethral bleeding, urinary retention. Our last comment addresses the fact that the study included 285 patients and 288 penile fractures, as 3 patients had a recurrent fracture-at the same site as the first one. This finding calls to attention that changes in the biomechanical properties of the tunica albuginea at the site of injury predosposes the organ to repeat fracture. The last consideration regards the patient's behavior during the sexual act. Since the persistence of the sexual behavior that caused the fracture may lead to a recurrent fracture, one can conjecture that the sparseness of recurrent fractures may stem from modifications in sexual behavior-decreases in the frequency of intercourse, avoidance of certain positions, for ins
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