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The spatial vectorcardiograms (sVCG) and electrocardiogramns were studied with respect to the gross and microscopic data in 59 instances of myocardial infarction. Besides the early abnormalities in the QRS sf-loop frequently associated with the electrocardiographic Q of infarction, there were recognizable alterations in the sVCG associated with the anatomic location of the infaretion: lateral wall infarets shortened vectors in iiid-QRS sfi-loop, and posterior lateral basal infarction altered the terminal portion distinctively. The myocardial lesions included fresh infarets, solid scars, scattered fibrosis, and scar with interwoven intact muscle fibers. Where many normal-appearing fibers were present in the lesion, the previously recorded ECG or sVCG frequently failed to show the diagnostic signs of infaretion. However, by presenting the depolarization complex in greater detail, the sVCG supplemented the ECG, improving the accuracy of diagnosis of infarction, especially among the smaller, less solid lesions.F OR several years it has been postulated1' and suggested by clinical studies3-that the spatial configuration of the vectoreardiogram would be altered in a predictable fashion by myocardial infarction and that these alterations would have some diagnostic significance. As theoretic considerations of electrocardiographic studies had suggested, the early portions of the QRS sE-loop may be visualized as spatially oriented away from the centroid of the infareted area and toward the centroid of the heart. Previous reports6' 9 have been based almost entirely upon clinical data; only 3 or 4 hearts7' were ever examined at necropsy, usually several weeks to years after recording of the spatial vectoreardiogram. The present investigation was undertaken 2 years ago to study the relation of the electrocardiogram (ECG) and the spatial vectorcardiogram (sVCG) to the clinical

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