Intracranial xanthogranulomas are rare benign tumors and occur most commonly as choroid plexus xanthogranulomas, which rarely become symptomatic. Exact etiology is still a matter of debate. We present a case of xanthogranuloma of the choroid plexus of the third ventricle, causing obstructive hydrocephalus of the lateral ventricles.

HE incidence of intracranial aneurysms in the general population is estimated to be less than 2%. a,14,16 Aneurysms occurring below the age of 20 years are quite uncommon. 1-3 McDonald and Korb, 12 in their collected series of 1125 intracranial aneurysms, noted that the youngest patient was 1 89 years old. They had only 28 patients younger than 15 years of age. Similarly, Taveras and Wood 17 found only four cases of intracranial aneurysms in patients under 15 years of age at the New York Neurological Institute during a period of 14 years. Only a few cases of aneurysms in early childhood have been reported2 ,7,11 The intracavernous aneurysms account for about 3% of all intracranial aneurysms2 In a country where the incidence of aneurysms is considered to be low, ~~ the occurrence of an intracavernous aneurysm of the internal carotid artery in a child aged 14 months is certainly a curiosity. Apparently the spontaneous cure of an aneurysm of the internal carotid artery in a young child has not been reported.

SUMMARY Two patients presenting with a clinical picture of nerve root compression suggestive of the herniated disc syndrome are reported. In both the diagnosis proved to be chronic encapsulated extradural haematoma. Recognition of this entity at the time of surgical exposure should render the surgical dissection easier. The postoperative results in both were excellent. One of these patients was on chronic anticoagulant therapy and the unusual location and good prognosis of extradural haematoma in the lumbar spinal canal in such patients is emphasized. In the other, the haematoma developed after a minor back injury, which introduces medico-legal implications.The occurrence of spontaneous haemorrhage in the spinal extradural space, though infrequent, is now a classifiable pathological entity.

W 'HILE it is not uncommon for a choroid plexus papilloma arising in the fourth ventricle to extend into the cerebellopontine angle, primary papilloma in this region is very rare2 ,~ In a detailed review of the literature, Morello and Migliavaca 4 found only nine such cases and added two cases of their own. Since then, to our knowledge, there has been no further report on primary choroid plexus papilloma in the cerebellopontine angle. This is a report of a primary cerebellopontine angle choroid plexus papilloma which was found at postmortem.Left-sided cerebellar and right-sided and cortico-spinal tract signs were also present. The routine laboratory examination was normal. Lumbar puncture revealed a pressure of 180 mm of water, 130 mg% of protein, and 45 mg% of sugar with no cells. Plain x-ray skull films did not reveal any abnormality. A right brachial angiogram was normal. Operation. A left suboccipital craniectomywas performed, and a partly cystic fairly vascular granulomatous extradural mass measuring 2.5 )< 1.5 cm was encountered. The dura appeared to be thickened. Palpa- Case ReportThis 34-year-old man was first seen in the neurological service on August 27, 1964, for left-sided facial weakness of 6 months' duration. He also complained of left-sided tinnitus, and diminished hearing. At that time, neurological examination revealed a lower motor neurone left facial weakness, left nerve deafness, and a nonfunctioning labyrinth on the left side. The patient refused admission.Examination. On December 8, 1966, the patient was admitted with difficulty in speech and swallowing. He was drowsy but cooperative, and was oriented in time and space. There was marked dysarthria. The disc margin on the right side was blurred, and the left fundus could not be seen because of severe kerato-conjunctivitis. There was bilateral lateral rectus palsy, left fifth cranial nerve palsy (both sensory and motor), left lower motor neurone facial weakness, left nerve deafness, weakness of palatal movement on the left side, and profuse discharge from the left ear.