The relationship between exercise and anxiety has been extensively examined over the last 15 years. Three separate meta-analysis were conducted to quantitatively review the exercise-anxiety literature for state anxiety, trait anxiety and psychophysiological correlates of anxiety. Such a procedure allows tendencies of the research to be characterised. The results substantiate the claim that exercise is associated with reductions in anxiety, but only for aerobic forms of exercise. These effects were generally independent of both subject (i.e. age and health status) and descriptive characteristics. Numerous design characteristics were different, but these differences were not uniform across the 3 meta-analyses. For state anxiety, exercise was associated with reduced anxiety, but had effects similar to other known anxiety-reducing treatments (e.g. relaxation). The trait anxiety meta-analysis revealed that random assignment was important for achieving larger effects when compared to the use of intact groups. Training programmes also need to exceed 10 weeks before significant changes in trait anxiety occur. For psychophysiological correlates, cardiovascular measures of anxiety (e.g. blood pressure, heart rate) yielded significantly smaller effects than did other measures (e.g. EMG, EEG). The only variable that was significant across all 3 meta-analyses was exercise duration. Exercise of at least 21 minutes seems necessary to achieve reductions in state and trait anxiety, but there were variables confounding this relationship. As such, it remains to be seen what the minimum duration is necessary for anxiety reduction. Although exercise offers therapeutic benefits for reducing anxiety without the dangers or costs of drug therapy or psychotherapy, it remains to be determined precisely why exercise is associated with reductions in anxiety. Since several mechanisms may be operating simultaneously, future research should be designed with the idea of testing interactions between these mechanisms.
Studies attempting to ascertain the effects of acute and chronic exercise on measures of sleep have yielded conflicting results and interpretations. Methodological differences among studies may explain this lack of consensus; however, small sample sizes and subsequently low statistical power may also have contributed. In an attempt to resolve these issues, this review used meta-analytical techniques to: (a) re-examine the effects of exercise on sleep; and (b) examine possible moderators of these effects. Studies meeting the selection criteria were included in the analysis. Analyses of moderating factors were performed for stage 4 sleep and rapid eye movement (REM) sleep. The results indicated that acute and chronic exercise increased slow wave sleep (SWS) and total sleep time but decreased sleep onset latency and REM sleep. Moderating variables influencing the magnitude and direction of these effects were related to characteristics of the individual (e.g. sex, age, fitness level) and the exercise (e.g. time of day exercise was completed, type of exercise, exercise duration). Mechanisms which have been suggested to explain the relationship between exercise and sleep are discussed and directions for further research are provided.
Mental fatigue was associated with increased power in frontal theta (θ) and parietal alpha (α) EEG rhythms. A statistical classifier can use these effects to model EEG-fatigue relationships accurately. Participants (n = 22) solved math problems on a computer until either they felt exhausted or 3 h had elapsed. Pre-and post-task mood scales showed that fatigue increased and energy decreased. Mean response times rose from 6.7 s to 7.9 s but accuracy did not change significantly. Mean power spectral densities or PSDs of θ and α bands rose by 29% and 44%, respectively. A kernel partial least squares classifier trained to classify PSD coefficients (1-18 Hz) of single 13-s EEG segments from alert or fatigued task periods was 91% to 100% accurate. For EEG segments from other task periods, the classifier outputs tracked the time course of the development of mental fatigue. By this measure, most subjects became substantially fatigued after 60 min of task performance. However, the trajectories of individual classifier outputs showed that EEG signs of developing fatigue were present in all subjects after 15-30 minutes of task performance. The results show that EEG can track the development of mental fatigue over time with accurate updates on a time scale a short as 13 seconds. In addition, the results agree with the notion that growing mental fatigue produces a shift away from executive and attention networks to default mode and is accompanied by a shift in alpha frequency to the lower alpha band.
Previous sport research on elite athletes has shown systematic changes in psychophysiological measures, such as heart rate (HR) deceleration and hemispheric asymmetries in EEG activity, in the few seconds prior to executing a motor response. These changes are believed to be due to a more focused attention on the external environment. Using archery (an attentive state), this investigation was designed to examine: (a) whether hemispheric asymmetry and HR deceleration would occur during the aiming period, and (b) if they did, whether this would affect performance. HR and left and right temporal EEG were recorded from 28 right-handed elite archers for 16 shots. The results indicated that (a) there was no HR deceleration; (b) during the aiming period, EEG alpha activity formed the dominant frequency and this was significantly greater in the left than in the right hemisphere; (c) there were no significant right hemisphere EEG changes in spectral power from 3 s before the shot to arrow release, but there were significant left hemisphere increases at 10, 12, and 24 Hz; and (d) at 1 s prior to the shot, there were no significant right hemisphere spectral power differences between best and worst shots, but there were significant left hemisphere differences at 6, 12, and 28 Hz. The relationships among hemispheric asymmetry, HR deceleration, attentional processes, and shooting performance are discussed.
scite is a Brooklyn-based organization that helps researchers better discover and understand research articles through Smart Citations–citations that display the context of the citation and describe whether the article provides supporting or contrasting evidence. scite is used by students and researchers from around the world and is funded in part by the National Science Foundation and the National Institute on Drug Abuse of the National Institutes of Health.
customersupport@researchsolutions.com
10624 S. Eastern Ave., Ste. A-614
Henderson, NV 89052, USA
This site is protected by reCAPTCHA and the Google Privacy Policy and Terms of Service apply.
Copyright © 2024 scite LLC. All rights reserved.
Made with 💙 for researchers
Part of the Research Solutions Family.