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The aim of this work is to investigate the possibility of heat insulation by "Temperature Swing", that is temperature fluctuation, on combustion chamber walls coated with low-heat-conductivity and low-heat-capacity materials.Adiabatic engines studied in the 1980s, such as ceramic coated engines, caused constantly high temperature on combustion wall surface during the whole cycle including the intake stroke, even if it employed ceramic thermal barrier coating methods. This resulted in increase in NOx and Soot, decrease in volumetric efficiency and combustion efficiency, and facilitated the occurrence of engine knock.On the other hand, "Temperature Swing" coat on the combustion chamber walls leads to a large change in surface temperature. In this case, the surface temperature with this insulation coat follows the transient gas temperature, which decreases heat loss with the prevention of intake air heating, and also which is expected to prevent NOx and Soot from increasing. In our calculations, the increase of the surface temperature fluctuation, "Temperature Swing" results from the coat of lower heat conductivity and lower heat capacity. Particularly in Gasoline engines, the coat with the appropriate thickness can reduce the heat flux from the wall to the working gas during intake stroke and can avoid engine knock.Based on our calculations, it is clarified that both the prevention of intake air heating and the low heat rejection were successfully possible with the material of appropriate thermo-physical properties.In addition to the calculations, a preliminary test-piece experiment was executed. It was demonstrated that the surface temperature of a porous coat, that is a candidate of "Temperature Swing" coat, immediately follows the transient gas temperature, and also the proposed insulation coat can exactly reduce the heat flux in the single-cylinder engine.

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Pancreatic secretory trypsin inhibitor (PSI!) has been thought to be only a secretory trypsin inhibitor of human pancreas, but the serum content of immunoreactive PSI! is elevated without pancreatic disease. Using the peroxidase-antiperoxidase method, immunoreactive cells for PSI! were found in human pancreas, stomach, duodenum, appendix, colon and urinary tract of both fetus and adult, adult gall bladder, and fetal lung. PSII-immunoreactive cells were identified in fetal pancreas at the tenth gestational week, and in extrapancreatic tissues at the sixteenth (gastrointestinal and urinary tract) and twentieth weeks (lung). PSTI-immunoreactive cells of fetal lung were present in neuroepithelial bodies.

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