The 1978 Kaysville, Utah, trench excavated by Swan and others (1980) across a large graben of the Weber segment of the Wasatch fault zone was reexcavated in 1988 to reevaluate the timing and nature of Holocene faulting. Relogging of the trench reveals evidence for five or six faulting events younger than the Provo phase of Lake Bonneville (circa 13,000 14C years B.P.). Geometric reconstruction of net vertical offset in the last three events suggests a variation in coseismic vertical displacement at this site, ranging from a net of 1.4–3.4 m per event. The three latest faulting events occurred at shortly before 0.6–0.8 ka, 2.8 ± 0.7 ka, and circa 3.8–7.9 ka. Earlier events cannot be directly dated because older graben‐fill sediments yielded thermoluminescence ages older than the time of deposition, and some scarp‐derived colluvial wedges beneath the trench floor were not exposed. The two younger faulting events we recognize at Kaysville correlate reasonably well with faulting events on the same segment 25 km north near East Ogden, Utah, at circa 0.8–1.2 ka and 2.5–3.0 ka (Forman and others, 1991), whereas the earlier Kaysville event is significantly older than the earliest (3.5–4.0 ka) event dated at East Ogden. The 3.5–4.0 ka ground rupture recognized at East Ogden may have died out at a subsegment boundary between the two trench sites within the 61‐km‐long Weber segment.

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The Utah Department of Health currently groups African-born blacks with U.S.-born blacks when reporting HIV/AIDS surveillance data. Studies suggest that categorizing HIV/AIDS cases in this manner may mask important epidemiological trends, and the distinct differences between these two populations warrant disaggregating data prior to reporting. The purpose of this study was to characterize the HIV/AIDS positive populations in U.S. and African-born blacks in Utah and evaluate the need for disaggregating the two groups. A total of 1,111 cases were identified through the statewide electronic HIV/AIDS Reporting System from 2000 - 2009. Data were analyzed for prevalence of HIV diagnosis for African-born blacks, U.S.-born blacks, and U.S.-born whites. Secondary analysis included HIV diagnosis by age, sex, African region of nativity, transmission risk factors, and differences in late diagnosis of HIV infection. U.S.-born whites accounted for 914 (82.3%) cases, and had the lowest annual prevalence (4/100,000). Conversely, African-born and U.S.- born blacks had the highest prevalence, 162/100,000 and 24/100,000 respectively. African-born blacks made up 0.25% of the total population, but accounted for 7.9% of all HIV/AIDS cases. African-born black males were more likely to report “no reported risk” for HIV transmission than U.S.-born black males. Of African-born blacks, 55.7% reported East-African nativity. These results demonstrate the importance of stratifying the black/African American racial category by African-born and U.S.-born blacks when collecting and reporting HIV/AIDS state surveillance data even in a low-incidence state,which will better inform prevention and linkage-to-care efforts in Utah.

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