BACKGROUND: Adolescence is a phase of rapid growth and increased nutritional needs. It includes the stressful burden of pubertal changes, both physically and psychologically. Moreover, it is associated with the utmost need for independence and identity formation. An adolescent finds a great way to practice taking their own decisions by making personal food choices. But unfortunately, wrong dietary choices lead to unsatisfactory nutritional status. AIM: To investigate the prevalence of six eating behaviours among adolescents. SUBJECTS AND METHODS: A case-control study was conducted on ninety Egyptian adolescents from 10 to 18 years old. Anthropometric measurements were taken. Body Mass Index (BMI) was calculated. The cases were forty-five children with body mass index ≥ 85th percentile. The control group involved forty-five of matched peers with body mass index ˂ 85th centile. A questionnaire form was constructed according to local customs in Egypt. RESULTS: Two unhealthy behaviours were mostly found in our study group. The first and the predominant one was multitasking while eating practised by 92.1% of candidates and showing the equal distribution in both groups. The second was skipping breakfast and was adopted by 51.7% of the study group with a significantly higher distribution in the ˂ 85th centile group. CONCLUSION: Faulty eating is a behaviour encountered in adolescence irrespective to BMI category. Thus, a normal BMI does not reflect healthy dietary behaviours.
BACKGROUND:Body weight concern and belief in adolescent females are of great importance. They are the keys to successful dietary interventions including dietary habits’ modifications to practice a healthy diet. This critical phase of transition from childhood to adulthood is the most sensitive stage of behavioural rectification.AIM:This study was conducted with the aim to figure out the prevalence of body image dissatisfaction and the association of body image satisfaction and believe with body mass index in adolescent girls aged 16-18 years.MATERIAL AND METHODS:Two hundred and three Egyptian adolescent females were enrolled in this cross-sectional study. Their mean age was (17.4 ± 0.64) years old. Self-administered questionnaires about the students’ body satisfaction and weight belief were answered by the candidates. Their body mass index was calculated. Also, sociodemographic data were collected. Data were analysed using SPSS software version 16.0. Chi-square test was conducted for the variables.RESULTS:Sixty-eight percent of the students were within normal weight, 3.3% were underweight, while 18.2% and 10.5% were overweight and obese respectively. Body dissatisfaction was prevalent among 37.4%. The prevalence of body dissatisfaction was higher in both obese and underweight candidates reaching (93.8% and 80%) respectively. This reflects students’ awareness of their body shape.CONCLUSION:More than one-third of adolescent females were dissatisfied with their body image. The subjective belief about self-body image matched the objective Body Mass Index measurements.
AIM:There are no reports regarding the influence of vitamin D on thymosin ß4 and the cluster of differentiation CD4 levels which are important for maintaining a healthy immune system. Consequently, we aimed to explore this relationship through a study.MATERIAL AND METHODS:The study was carried out on 35 subjects, screened for 25-hydroxy vitamin D[25 (OH) D] using ELISA method and they were divided into two groups: Group 1 consists of 10 healthy subjects with sufficient vit. D level > 24.8 ng/ml. Group 2 consists of 25 subjects suffering, severely, from vitamin D deficiency at level < 11.325 ng/ml. Also, Thymosin ß4, CD4 and zinc levels were performed.RESULTS:There were significant differences between the two groups in the concentration levels of thymosin β4, as the group 1 has shown higher levels (P = 0.005). Whereas, CD4 and zinc levels didn’t show any significant difference between the two groups. At the same time, a significant positive correlation has been observed between vitamin D, thymosin β4, and CD4 at (r = 0.719; P = 0.001), and (r = 0.559, P = 0.001) respectively.CONCLUSION:We concluded that vitamin D may be an essential factor that influence or determine the level of thymosin β4. This study is the first that focused on demonstrating that sufficient level of vitamin D may have the ability to influence the thymic hormone thymosin β4 levels. Further studies on large scale of subjects are needed to explore the positive correlation we had found between vitamin D and thymosin β4 and CD4.
Our study concludes that mutations of NPHS2 gene are common among Egyptian children with SRNS. We support a model where ethnicity plays an important role in specific NPHS2 mutations, since a novel mutation was found in one patient in this study. Future study on a large number of Egyptian patients with SRNS is warranted to identify the actual genetic contribution of this gene in the development of SRNS in our population, which might help in patients' prognosis and management.
Vitamins are evaluated for their role in immunity. Recently, vitamin A received a particular attention as a critical micronutrient for regulating immune system. Therefore, the present study aimed to search for new about vitamin A. Forty-eight Egyptian adults aged from 18 to 42 years old from both sexes were subjected to clinical examination and nutrition questionnaire and were screened for vitamin A by using ELISA method. Forty subjects were selected and subdivided into two groups. Group 1 with vitamin A at level >200 µg/dl consists of 10 healthy subjects. Group 2 with vitamin A deficiency at level <50 µg/dl consists of 30 subjects. Tβ4 and CD4 levels were also determined by a commercial ELISA kit. Results showed a significant decrease in serum levels of Tβ4 and CD4 in group 2 than group 1 at P < .003 and P < .019 respectively. Both of Tβ4 and CD4 had positive correlation with vitamin A level at P < .000 and P < .003 respectively as well as with each other at p < .000. We concluded that vitamin A deficiency may be influence the levels of Tβ4 and CD4.
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