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increase in androgen receptor levels or their distribution between lesional and normal skin. In the present study, the expression of androgen receptors within lesional skin was not increased. Our findings cannot rule out the possibility of an endocrine influence at the microvascular level and we believe that unopposed androgenic stimulation might still contribute to the pathogenesis of rosacea. 3 Feldman SR, Hollar CB, Gupta AK, Fleischer AB Jr. Women commonly seek care for rosacea: dermatologists frequently provide the care. Cutis 2001; 68 : 156 -160. 4 Thibout DM. Acne rosacea. Am Fam Physician 1994; 50 : 1691-1697. 5 Schmidt JB, Raff M, Spona J. Do sexual steroids contribute to the pathomechanism of rosacea? A study of estrogen and androgen receptors in acne rosacea. Acta Derm Venereol (Stockh) 1983; 63 : 64 -66. 6 Spirov G, Berova N, Vassilev D. Effect of oral inhibitors of ovulation in treatment of rosacea and dermatitis perioralis in women. Aust J Dermatol 1971; 12 : 149 -154. 7 Zuber TJ. Rosacea. Primary Care 2000; 27 : 309 -318. 8 Mauss J. Cyproterone therapy of papulopustular rosacea in women. Hautarzt 1981; 32 : 94 -95. 9 Koulinas GT. Treatment of acne with oral contraceptives: criteria for pill selection. Cutis 2000; 66 : 281-286. 10 McPhaul MJ, Young M. Complexities of androgen action.

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