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Acute and chronic sleep loss are linked with a range of negative physiological and psychological outcomes (Kecklund & Axelsson, 2016). While complete sleep deprivation rapidly impedes simple and complex cognitive functions, sleep restriction impairs whole-body homeostasis, leading to undesirable metabolic consequences in the short-and longer-term (Reutrakul & Van Cauter, 2018). Most metabolic tissues including liver (Shigiyama et al., 2018), adipose tissue (Wilms et al., 2019), and skeletal muscle are at risk of developing sleep loss-associated adverse outcomes.Skeletal muscle is a primary regulator of human metabolism. Sleep deprivation (Cedernaes et al., 2015(Cedernaes et al., , 2018 and restriction (Harfmann et al., 2015) have the potential to profoundly affect muscle health by altering gene regulation and substrate metabolism. Even relatively short periods of sleep restriction (less than a week) can compromise

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