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Uterine arteriovenous malformations (AVMs) are a rare pathology. They comprise a large spectrum of lesions that are classified in accordance with their content and flow. Most cases are diagnosed during an obstetric hemorrhagic event. Patients are usually asymptomatic until the occurrence of massive transvaginal bleeding. The endovascular approach is currently preferred and management will depend on the type of AVM, sclerotherapy, embolization, or surgery. We present the case of a healthy 22-year-old patient with a history of intrauterine curettage 2 months before her hospitalization due to a miscarriage. Begins with 20 days of evolution with abnormal vaginal bleeding in treatment with steroids without improvement. Ultrasound with the presence of AVM in the anterior wall of the uterus. The patient underwent endovascular embolization by transcatheter with microparticles with subsequent remission of metrorrhagia.

Introduction: Thoracic outlet syndrome (TOS) results in the compression of neurovascular structures (brachial plexus, subclavian artery, and vein) between the scalene muscles and first rib. Compression of these structures can result in neurogenic and vascular TOS (arterial and venous). Case Report: We present the case of a 40-year-old female patient with acute upper limb ischemia secondary to arterial TOS. Discussion: Arterial TOS is a rare cause of ischemia of the upper limbs. It is the least common form of TOS, which represents < 1% of TOS cases. However, arterial TOS is potentially dangerous, as it can lead to acute ischemia of the upper extremities, damage of the intima, and aneurysmal degeneration of the subclavian artery related to chronic compression. Knowledge of the arterial TOS is crucial to reach a timely diagnosis and management.

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