Peritonitis is a very serious complication encountered in patients undergoing peritoneal dialysis and healthcare providers involved in the management should be very vigilant. Gram-positive organisms are the frequent cause of peritonitis compared to gram-negative organisms. There has been recognition of peritonitis caused by uncommon organisms because of improved microbiological detection techniques. We report a case of peritonitis caused by Moraxella osloensis (M. osloensis), which is an unusual cause of infections in humans. A 68-year-old male, who has been on peritoneal dialysis for 2 years, presented with abdominal pain and cloudy effluent. Peritoneal fluid analysis was consistent with peritonitis and peritoneal fluid culture grew gram-negative bacteria. M. osloensis was identified by 16 S PCR phenotypic and sequencing techniques. Patient responded well to the treatment, with intraperitoneal cephalosporin, and repeat peritoneal fluid culture yielded no growth. M. osloensis rarely causes infection in humans and responds well to treatment, as reported in literature.
We present a case of hypercalcemia in a 79-year-old female likely secondary to uterine leiomyoma. To the best of our knowledge, hypercalcemia due to a benign tumor has only been described in five cases. Of these above five cases, uterine leiomyoma was thought to be the cause of hypercalcemia in three cases.
Yersinia enterocoliticais primarily a gastrointestinal tract pathogen known to cause gastroenteritis, although it may produce extra-intestinal infections like sepsis and its sequelae. However, primary cutaneous infections are extremely rare. We present a case ofY. enterocoliticathigh abscess in an immunocompetent adult. The portal of entry is unclear in this case. He did many outdoor activities that involved skin injuries and exposure to soil and contaminated water. Hence, direct inoculation as a result of exposure to contaminated water is postulated in the absence of evidence for a gastrointestinal route of infection.
sistent with the findings of a previous study 8 that found no statistically significant differences in plasma total bilirubin levels between individuals with AD (n = 143) and cognitively healthy controls (n = 1,553), although another study 9 found significantly lower plasma bilirubin levels in individuals with AD (n = 101; 0.6 AE 0.2 mg/ dL) than in cognitively healthy controls (n = 101; 0.9 AE 0.2 mg/dL) (P < .001), and a third study 10 found that of CSF bilirubin levels were significantly higher (P < .001) in individuals with AD than in controls. Mean CSF unconjugated bilirubin level was not significantly different between the groups. Additional larger prospective studies are needed to evaluate the role of plasma bilirubin as a biomarker in individuals with AD.
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