Growing concerns about the vulnerability of the electric grid, uncertainty about the cost of oil, and an increase in the deployment of renewable generation on domestic military installations have all led the Department of Defense (DoD) to reconsider its strategy for providing energy security for critical domestic operations. Existing solutions typically use dedicated backup generators to service each critical load. For large installations, this can result in over 50 small generators, each servicing a low voltage feeder to an individual building. The system as a whole is typically not well integrated either internally, with nearby renewable assets, or to the larger external grid. As a result, system performance is not optimized for efficient, reactive, and sustainable operations across the installation in the event of a power outage or in response to periods of high stress on the grid. Recent advances in energy management systems and power electronics provide an opportunity to interconnect multiple sources and loads into an integrated system that can then be optimized for reliability, efficiency, and/or cost. These integrated energy systems, or microgrids, are the focus of this study. 4. ENVIRONMENT 4.1 Location-dependent characteristics (resources and weather) 4.2 Domestic electric grid characteristics 4.3 Department of Defense installation characteristics 5. MODELING AND ANALYSIS 5.1 Description of microgrid architectures 5.2 Analysis methodology 5.3 Analysis inputs 5.4 Analysis results 6. RECOMMENDATIONS

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This study aimed to assess the viability of dental cells following time-dependent carbamide peroxide teeth-whitening treatments using an in-vitro dentin perfusion assay model. 30 teeth were exposed to 5% or 16% CP gel (4 h daily) for 2-weeks. The enamel organic content was measured with thermogravimetry. The time-dependent viability of human dental pulp stem cells (HDPSCs) and gingival fibroblast cells (HGFCs) following either indirect exposure to 3 commercially available concentrations of CP gel using an in-vitro dentin perfusion assay or direct exposure to 5% H2O2 were investigated by evaluating change in cell morphology and by hemocytometry. The 5% and 16% CP produced a significantly lower (p < 0.001) enamel protein content (by weight) when compared to the control. The organic content in enamel varied accordingly to the CP treatment: for the 16% and 5% CP treatment groups, a variation of 4.0% and 5.4%, respectively, was observed with no significant difference. The cell viability of HDPSCs decreased exponentially over time for all groups. Within the limitation of this in-vitro study, we conclude that even low concentrations of H2O2 and CP result in a deleterious change in enamel protein content and compromise the viability of HGFCs and HDPSCs. These effects should be observed in-vivo.

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