・Of 104 cases analyzed, 76 (73%) were asymptomatic, 41 (54%) of which had pneumonic changes on CT. Other 28 (27%) cases were symptomatic, 22 (79%) of which had abnormal CT findings. ・Asymptomatic cases showed more GGO predominance over consolidation (83%), while symptomatic cases were more likely to show a consolidation predominance over GGO (41%). ・Asymptomatic cases showed milder CT severity score than symptomatic cases.

This erratum corrects an error in the number of cases in the study. The number of cases decreased from 112 to 104. These eight cases need to be omitted from the study for the following reasons:After submission of the manuscript, three persons withdrew their consent for research on discharge. As per IRB approval documentation of the study, three people need to be omitted from the study. Authors were notified about their withdrawal from the IRB only after online publication.The authors double-counted five cases because they had different names, and different study numbers were assigned. Many of the passengers of the cruise ship spoke neither English nor Japanese, and the names they were given at the initial triage on admission later proved to not be the correct ones. The PACS system recorded these persons as different cases for their follow-up study, and the study number was differently assigned for those cases.The authors conducted calculations and statistical analysis for these 104 cases and corrected the numbers and percentages in the Results section. Other parts including Conclusion were not changed.These changes were made online on April 8, 2020. Data changes are in bold.In the first two Key Points, Of 104 cases analyzed, 76 (73%) were asymptomatic, 41 (54%) of which had pneumonic changes on CT. Other 28 (27%) cases were symptomatic, 22 (79%) of which had abnormal CT findings. Asymptomatic cases showed more GGO predominance over consolidation (83%), while symptomatic cases were more likely to show a consolidation predominance over GGO (41%).Abstract, Materials and Methods, This retrospective study comprised 104 cases (mean age, 62 years ± 16, range 25-93) with COVID-19 confirmed with RT-PCR.Abstract, Results, Of 104 cases, 76 (73%) were asymptomatic, 41 (54%) of which had lung opacities on CT. Other 28 (27%) cases were symptomatic, 22 (79%)

Binding of the purified type C 75 (neurotoxin), 125 and 165 botulinum toxins to epithelial cells of ligated small intestine or colon of the guinea pig (in vivo test) and to pre-fixed gastrointestinal tissue sections (in vitro test) was analysed. The 16s toxin bound intensely to the microvilli of epithelial cells of the small intestine in both in vivo and in vitro tests, but did not bind to cells of (2 x lo5 MLD) was injected, little toxin activity was detected in the sera.Therefore, the haemagglutinin of type C 16s toxin is apparently very important in the binding and absorption of botulinum toxin in the small in test i ne.1

A "Honda sign or variation" with evidence of fractures elsewhere or no evidence of other metastatic disease should be strong evidence for a sacral insufficiency fracture. The likelihood of having a solitary metastasis to the sacrum is small.

The effects of exercise and catecholamines on platelet reactivity or coagulation and fibrinolysis appear to be inconsistent. This may be partly due to the methods employed in previous studies. In the present study, we investigated the effects of acute aerobic exercise and catecholamines on the thrombotic status by a novel in vitro method, shear-induced hemostatic plug formation (hemostatometry), using nonanticoagulated (native) blood. Aerobic exercise (60% maximal O2 consumption) was performed by healthy male volunteers for 20 min, and the effect on platelet reactivity and coagulation was assessed by performing hemostatometry before and immediately after exercise. Exercise significantly increased shear-induced platelet reactivity, coagulation, and catecholamine levels. The effect of catecholamines on platelet reactivity and coagulation was assessed in vitro by adding catecholamines to blood collected in the resting state. The main findings of the present study are that elevation of circulating norepinephrine at levels that are attained during exercise causes platelet hyperreactivity and a platelet-mediated enhanced coagulation. This may be a mechanism of an association of aerobic exercise with thrombotic risk.