Simple renal cyst (SRC) is uncommon paediatric pathology. It's spherical, round to oval. Perceptible wall, anechoic, without septa, and cyst is separated from the collecting system, and the cyst has no colour Doppler flow. They may be as small as a pea or as large as a golf ball. Simple renal cysts are filled with serous fluid and have a simple epithelial covering. They are less common in children with incidences of less than 0.5 percent, while adults have incidence of more than 10 %, and more than 30 % in adults aged 70 and older. The exact mechanism by which renal cysts form is unknown. There are a few theories of how the disease develops. The causative factor, according to one hypothesis, is a focal ischemia in a renal tubule that causes localised obstruction. According to another hypothesis, renal cysts are caused by calyceal diverticula that have lost contact with the adjacent calyx.1 Because of the increased use of ultrasound screening (USS), more children are being diagnosed with renal cysts. Asymptomatic and symptomatic cysts (abdominal pain, hematuria, etc.) are distinguished, but mostly asymptomatic cysts develop slowly, though complications such as bleeding, infection, or rupture may occur, particularly in younger children.2 Chronic kidney disease (CKD), polycystic kidney disease, malignant transformation, and symptomatic enlargement have all been identified in paediatric patients with renal cysts. The vast majority of renal cysts, fortunately, do not develop and remain largely unchanged over time. In cases of asymptomatic simple renal cysts in infants, a conservative management approach is used. Recent research supports the use of an ultrasound-based (US) adapted Bosniak classification system to classify renal cysts in children. This would result in lower rates of surgical treatments for benign lesions, as well as less ionising radiation from needless computed tomography (CT) scans. It would also mean less extended monitoring and associated morbidity.

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Non-Hodgkins lymphoma involves lymph nodes which show a characteristic micronodular (reticulated) pathognomonic pattern on sonography. We are presenting sonography, Doppler, and strain elastography findings with pathological correlation in a 65-year-old patient with posterior neck lymphadenopathy.

Mirror syndrome, by definition, is maternal edema with fetal hydrops. Majority of the pregnancies in developing countries are complicated by preeclampsia and intrauterine growth retardation. Fetal ascites, on the other hand, is a part of spectrum of the condition under hydrops fetalis. Abnormal umbilical artery Doppler is an indicator of fetal growth retardation and not many cases are found with such diversely intermixed conditions. A 26-year-old primigravida with 36 weeks gestation by the last menstrual cycle and 34 weeks by ultrasound presented to our hospital with a history of decreased fetal movements, headache, fever, weakness, dizziness, and new-onset raised blood pressure. Her blood pressure was 164/118 and 3+ proteinuria on the urinary dipstick test. A diagnosis of preeclampsia was made. An antenatal ultrasound was performed, which showed fetal ascites, mild thickening of fetal skin, oligohydramnios with reversed umbilical artery flow but normal uterine arteries, and middle cerebral arteries. The blood pressure was controlled and she later delivered a low-birth weight baby that needed neonatal intensive care unit stay for fetal distress and abdominal distention. The baby did not survive the coming few days and died due to increasing fluid in the body cavity. The mother continued to have raised blood pressure in the postpartum period. Mirror syndrome is when the maternal pathology mirrors that of the fetus. Preeclampsia, fetal hydrops, and placental edema are the conditions lead to suspicion of it. In the case, we present, the mother had preeclampsia, and there was fetal ascites and reversed umbilical flow. The fetal and maternal outcome was in the form of intrauterine growth retardation, impending hydrops fetalis and chronic hypertension, respectively.