Multi-core processors integrate with multiple computing units on one chip. This technology is increasingly mature, and communication between cores has become the largest research hotspot. As the number of cores continues to increase, the humble bus structure can no longer play the role of multi-core processors. Network on chip (NoC) connects components through routing, which greatly enhances the efficiency of communication. However, the communication power it consumes and network latency are issues that cannot be ignored. An efficient mapping algorithm is an effective method to reduce the communication power and network latency. This paper proposes a mapping method. First, the task is divided depending on the scale of the task. When the task scale is small, to reduce the communication distance between resource nodes, a given NoC substructure is selected to map the task; when the task scale is large, to reduce the communication between tasks, the tasks are clustered and tasks with dependencies are divided into the same resource node. Then combine with an improving ant colony algorithm (ACO) for mapping. The method proposed is being experimentally verified on NoC platforms of different scales. The experimental results show that the method proposed is very effectual for reducing communication power and network latency during NoC mapping.

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Liver cancer is a typical malignant tumor and the fourth most typical cancer in the Earth. Clinically, more than 90% of patients with primary liver cancer are caused by hepatocellular carcinoma. Epidemiological and experimental data show that human infection with HBV and HCV hepatitis virus is firmly connected with the occurrence of liver cancer. In the past 20 years, the level of diagnosis and cure of liver cancer in China has been greatly improved, but little progress has been made in the study of liver cancer markers. For then, with the understanding of Wnt signal transduction pathway, it is found that β-catenin, as a key molecule in Wnt signal transduction pathway, is closely associated to the incidence of HCC. Recently, more and more studies have shown that the abnormal activation of classical Wnt signal pathway plays a compelling part in the occurrence and advancement of hepatocellular carcinoma. Hepatocytes because of hidden onset, early rise can be no clinical symptoms, so the clinical discovery is mostly late, the mortality rate is high. At present, the clinical methods for the treatment of hepatocellular carcinoma are liver transplantation, radiotherapy and chemotherapy. However, hepatocellular carcinoma is easy to metastasize and has a high recurrence rate, so the mortality rate of hepatocellular carcinoma is still high. Wnt/β-catenin signal pathway has become major topic of debate in cancer research. In this paper, we begin with the classification of liver cancer and some pathogenic mechanisms. The effects of β-catenin protein on the occurrence, metastasis and immune regulation of hepatocellular carcinoma were also discussed. Under the background of the popular "signal transduction therapy" in recent years, to explore the therapeutic effect of targeted drugs targeting Wnt/β-catenin signal pathway in hepatocellular carcinoma. Because of the high degree of malignancy and limited treatment of hepatocellular carcinoma, we will mainly discuss the effect of Wnt signal pathway on the metastasis of hepatocellular carcinoma and its effect on the differentiation of immune cells. Abnormal activation of Wnt signaling pathway in hepatocellular carcinoma.