The fully nonlinear development of small-wavelength Görtler vortices in a growing boundary layer is investigated by a combination of asymptotic and numerical methods. The starting point for the analysis is the weakly nonlinear theory of Hall ( J. Inst. Math. Applies 29, 173 (1982)) who discussed the initial development of large-wavenumber small-amplitude vortices in a neighbourhood of the location where they first become linearly unstable. That development is unusual in the context of nonlinear stability theory in that it is not described by the Stuart-Watson approach. In fact, the development is governed by a pair of coupled nonlinear partial differential evolution equations for the vortex flow and the mean flow correction. Here the further development of this interaction is considered for vortices so large that the mean flow correction driven by them is as large as the basic state. Surprisingly it is found that such a nonlinear interaction can still be described by asymptotic means. It is shown that the vortices spread out across the boundary layer and effectively drive the boundary layer. In fact, the system obtained by the equations for the fundamental component of the vortex generates a differential equation for the basic state. Thus the mean flow adjusts so as to make these large amplitude vortices locally neutral. Moreover in the region where the vortices exist the mean flow has a ‘square-root’ profile and the vortex velocity field can be written down in closed form. The upper and lower boundaries of the region of vortex activity are determined by a free-boundary problem involving the boundary-layer equations. In general it is found that this region ultimately includes almost all of the original boundary layer and much of the free stream. In this situation the mean flow has essentially no relation to the flow that exists in the absence of the vortices.

No abstract

Idiopathic intracranial hypertension (IIH) is a syndrome of unknown etiology characterized by elevated intracranial pressure (ICP). Although a stenosis of the transverse sinus has been observed in many IIH patients, the role this feature plays in IIH is in dispute. In this paper, a lumped-parameter model is developed for the purpose of analytically investigating the elevated pressures associated with IIH and a collapsible transverse sinus. This analysis yields practical predictions regarding the degree of elevated ICPs and the effectiveness of various treatment methods. Results suggest that IIH may be caused by a sufficiently collapsible transverse sinus, but it is also possible that a stenosed sinus may persist following resolution of significant intracranial hypertension.

This work considers a Blasius boundary value problem with inhomogeneous

The experimentally-measured pressure-volume relationship for the human intracranial system is a nonlinear`S-shaped'curve with two pressure plateaus, a point of inflection, and a vertical asymptote at high pressures where all capacity for volume compensation is lost. In lumped-parameter mathematical models of the intracranial system, local compliance parameters relate volume adjustments to dynamic changes in pressure differences between adjacent model subunits. This work explores the relationship between the forms used for local model compliances and the calculated global pressure-volume relationship. It is shown that the experimentally-measured global relationship can be recovered using physiologically motivated expressions for the local compliances at the interfaces between the venouscerebrospinal fluid (CSF) subunits and arterial-CSF subunits in the model. Establishment of a consistent link between local model compliances and the physiological bulk pressurevolume relationship is essential if lumped-parameter models are to be capable of realistically predicting intracranial pressure dynamics.