The goal of this study is to see how cold plasma affects rabbit bone tissue infected with osteoporosis. The search is divided into three categories: control, infected, and treated. The rabbits were subjected to cold plasma for five minutes in a room with a microwave plasma voltage of “175 V” and a gas flow of “2.” A histopathological photograph of infected bone cells is obtained to demonstrate the influence of plasma on infected bone cells, as well as the extent of destruction and effect of plasma therapy before and after exposure. The findings of the search show that plasma has a clear impact on Ca and vitamin D levels. In the cold plasma, the levels of osteocalcin and alkali phosphates (ALP) respond as well. Image processing techniques (second-order gray level matrix) with textural elements are employed as an extra proof. The outcome gives good treatment indicators, and the image processing result corresponds to the biological result.
This research explores how e-health systems’ features (information quality, quality of the system, usability perceived, and perceived usefulness) contribute to improving medical personnel performance in medical centers, patient care, and physician-patient interactions in Jordan. The objective is to evaluate a single integrated model consisting of the technology acceptance model. This study used the logical research method and approach. A collection of data from 212 medical personnel working in 19 healthcare facilities throughout Jordan were gathered. To analyze the data collected and test the hypotheses of the research, a partially square/structural equation modeling method has been employed. The study found that the health information system (HIS) information quality has a direct and indirect beneficial effect on the performance of the staff, beneficial effects on patient care alone, and only favorable, indirect effects on the doctor-patient relationship. On the contrary, system quality was shown to influence directly and indirectly and to have a direct and indirect beneficial effect both on the connection between doctors and patients. Remember that the HIS has accessibility, speed, and mistake detection and avoids error issues. These shortcomings are suggested to be rectified in conjunction with improved user perception towards easy usage and utilization of the system.
The aim of this study is to demonstrate the effect of particle size on semiconductor properties; artificial intelligence is being used for the research methods. As a result, we picked cadmium sulfide (CdS), which is a unique semiconductor material that is employed in a broad variety of current applications. Given that CdS has distinct electrical and optical characteristics, it may be employed in the production of solar cells, for example. Solar cells, as is also well known, have become an essential source of energy in the world. Within the visible range (500-700 nm), we create one layer of bulk CdS and one layer of nano-CdS air bulk CdS air and air nano-CdS air. We used a number of instrumentation methods to investigate the naked CdS nanoparticles, including XRD, SEM-EDX, UV-Vis spectroscopy, TEM, XPS, and PL spectroscopy, among others. The results show that for bulk CdS at normal incidence, the transmittance is
T
=
45
, and for nano-CdS with particle size 3 nm, the transmittance is
T
=
85.8
, with transverse-electric (S-polarized) and transverse-magnetic (P-polarized) transmittances of
TE
=
75
and
TM
=
80
, respectively.
Liver cirrhosis results from prolonged and extensive liver fibrosis in which fibrotic tissues replace functional hepatic cells. Chronic liver disease due to various viral, chemical, or metabolic factors initiates hepatic fibrogenesis. Cirrhosis is associated with multiple clinical complications and a poor patient prognosis; therefore, developing novel antifibrotic therapies to prevent cirrhosis is of high priority. Mounting evidence points to the key role of serum response factor (SRF) and myocardin‐related transcription factor (MRTF)‐A in the pathogenesis of liver fibrosis. SRF is a transcription factor and MRTF‐A is a co‐activator of SRF and normally resides in the cytoplasm. Upon the induction of fibrotic pathways, MRTF‐A translocates into the nucleus and forms the active SRF/MRTF‐A complex, leading to the expression of a multitude of fibrotic proteins and components of extracellular matrix. Silencing or inhibiting MRTF‐A impedes hepatic stellate cell transdifferentiation into myofibroblasts and slows down the deposition of extracellular matrix in the liver, making it a potential therapeutic target. Here, we review the recent findings regarding the role of the SRF/MRTF‐A complex in liver fibrosis and its therapeutic potential for the management of cirrhosis.
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