Detecting forest fire smoke during the initial stages is vital for preventing forest fire events. Recent studies have shown that exploring spatial and temporal features of the image sequence is important for this task. Nevertheless, since the long distance wildfire smoke usually move slowly and lacks salient features, accurate smoke detection is still a challenging task. In this paper, we propose a novel Attention Enhanced Bidirectional Long Short-Term Memory Network (ABi-LSTM) for video based forest fire smoke recognition. The proposed ABi-LSTM consists of the spatial features extraction network, the Bidirectional Long Short-Term Memory Network(LSTM), and the temporal attention subnetwork, which can not only capture discriminative spatiotemporal features from image patch sequences but also pay different levels of attention to different patches. Experiments show that out ABi-LSTM is capable of achieving best accuracy and less false alarms on different types of scenarios. The ABi-LSTM model achieve a highly accuracy of 97.8%, and there is 4.4% improvement over the image-based deep learning model.

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Background Vascular remodeling is the most critical pathogenesis of atherosclerosis. Adipokine chemerin was known for its relationship with obesity as well as metabolism. Most recently, chemerin was found to play a crucial role in the pathologic process of cardiovascular diseases including coronary heart disease. In this study, we surveyed the role of chemerin in progression of atherosclerosis in ApoE−/− mice. Objective To investigate the relationship between chemerin and progression of atherosclerosis in ApoE−/− mice and its mechanism. Methods 8-week-old ApoE−/− mice were fed with high-fat diet to induce the atherosclerosis model. Adenoviruses were transfected for knockdown or overexpression of chemerin gene into aorta. Serums and aortic tissues of ApoE−/− mice were obtained after feeding high-fat diet for 16 weeks. HE staining and oil red staining were performed to evaluate aortic plaque. ELISA was performed to explore serum levels of tumor necrosis factor-α (TNF-α), interleukin-1β (IL-1β), and transforming growth factor-β1 (TGF-β1). Real-time PCR and western blotting were carried out to investigate the mRNA and protein levels of chemerin, nuclear factor-κB p65 (NF-κBp65), proliferating cell nuclear antigen (PCNA), phosphorylated p38 mitogen-activated protein kinase (p-p38-MAPK), phosphorylated c-Jun N-terminal kinase (p-JNK), and phosphorylated extracellular signal regulated kinase 1/2 (p-ERK 1/2). Result Aortic plaque formation was significantly induced by high-fat diet in ApoE−/− mice. Simultaneously, elevated serum levels of TNF-α and IL-1β and elevated mRNA and protein levels of chemerin, NF-κBp65, PCNA, p-p38-MAPK, p-JNK, and p-ERK 1/2 were found in ApoE−/− mice. After aortic chemerin gene was inhibited by adenovirus, aortic atherosclerosis induced by high-fat diet was significantly meliorated, serum levels of TNF-α and IL-1β decreased, mRNA and protein levels of NF-κBp65, PCNA, p-p38-MAPK, p-JNK, and p-ERK 1/2 decreased simultaneously. Conclusion Our study revealed that chemerin stimulated the progression of atherosclerosis in ApoE−/− mice.

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