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Although vitiligo is a multifactorial skin disease, accumulating data have strongly indicated that melanocytes are ultimately destroyed by a cascade of autoimmune responses. Contrast to widely accepted T cell-based cellular immunity, the role of B cell-based humoral immunity in vitiligo remains elusive. The present study investigated the changes of distributions and fucntions of circulating B cells and Tfh cells in patients with active non-segmental vitiligo. Compared with HC, the antibody