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This study aimed to examine the indicators of gum epithelial cell proliferation in patients with chronic periodontitis against gastroesophageal reflux disease (GERD). The clinical, endoscopic, immunohistological and morphological studies revealed that diseases affecting the esophagus facilitate the development and the progression of periodontal issues. Enhanced apoptosis due to insufficient proliferation of periodontal tissues contributes to the progression of inflammatory and destructive changes in the tissues, against the background of reduced regeneration due to a decrease in the Ki-67 immunopositive cells.

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s t o M a t o L o G Y C L i N i C a L r E s E a r C h 132 | a r c h i v e u r o m e d i c a | 2 0 1 9 | v o l . 9 | n u m . 2 | s t o M a t o L o G Y C L i N i C a L r E s E a r C h

No abstract