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In the accounting education process, theoretical -practical information is taught as a whole. In this period in which several teaching methods such as cooperative learning, discussion, case studies and so on, are used, the methods are being shaped as educational factor and just one or several of these methods can be used together. The learning style concept is defined in literature as an individual way chosen in analyzing and comprehending. The learning styles are innate and it affects the people's learning process. It is how a person learns than what a person learns. In this context, the students' success is related directly with the right learning style. So the aim of the study is measuring the influence of learning styles over the students taken accounting courses. Therefore a questionnaire will be applied to the students of ASU FEAS and the data collected will be analyzed with SPSS package program. With the results, we aim bringing out the inventory of students' learning styles and the performance relations.

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Although there are some studies suggesting relation between different types of infection and fibromyalgia syndrome (FM), there is presently no proof that FM is caused by an infection. Helicobacter pylori (HP) infection may cause extragastric manifestations. Inflammation is an important mediator of increased sympathetic nervous system activity and may lead to pain in fibromyalgia patients. In this study, we aimed to investigate the HP seropositivity in fibromyalgia patients compared with controls for possible role of HP infection in FM. Sixty-seven patients with fibromyalgia were evaluated. Two of them were excluded from the study because of high level of acute phase reactants. Sixty-five female patients with fibromyalgia and 41 randomly selected age-matched female healthy controls were enrolled to study. Serum HP IgA and IgG antibodies were measured by enzyme-linked immunosorbent assay technique. Fibromyalgia Impact Questionnaire was assessed in patients and controls. Seropositivity of HP IgG antibody in the fibromyalgia patients was significantly higher than in the control group. No statistically significant differences were found regarding the clinical features between fibromyalgia patients with HP IgG antibody and patients without IgG antibody. Our study suggests that former HP infection may have a role in the etiopathogenesis of fibromyalgia syndrome or may act as a triggering factor. However, high seroprevalence of HP in general population and prevalent asymptomatic infection make it difficult to interpret these results for the definite role of HP in FM. Highlighting the pathophysiologic mechanisms of FM will result in more effective treatment regimens.

Abstract.[Purpose] The results of studies investigating the role of Helicobacter pylori (HP) in osteoporosis are contradictory. In this study we investigated HP seropositivity in postmenopausal osteoporotic and non-osteoporotic females to elucidate the role of HP in postmenopausal osteoporosis. [Subjects and Methods] Serum was collected from fifty-eight osteoporotic patients and forty-seven nonosteoporotic subjects, whose status was diagnosed with dual-energy X-ray absorptiometry (DEXA). None of the subjects had received any prior treatment for osteoporosis. Subjects' sera were assessed for HP antibodies (Immunoglobulin A and G) by enzyme-linked immunoabsorbent assay (ELISA). Patients were interviewed about risk factors of osteoporosis. Prior fractures of patients and familial fracture history were also noted.[Results] Fifty-eight patients with osteoporosis and forty-seven nonosteoporotic patients, as determined by lumbar total or collum femoris in DEXA, were evaluated in this study. The familial fracture history was significantly higher in the osteoporotic patients than in the nonosteoporotic group. There was no significant difference in HP seropositivity between the osteoporotic and nonosteoporotic groups.[Conclusion] There was no difference in HP seropositivity between the groups, therefore HP infection seems not to be an important risk factor for postmenopausal osteoporosis.