Campylobacter were detected by PCR in feces of monkeys of different species (clinically healthy, with diarrhea, and dead from acute enteric infections). High prevalence of these bacteria in monkeys was revealed. The incidence of C. jejuni DNA in monkeys with acute enteric infections was higher than in healthy animals (69.6 and 51.3%, respectively). The highest percentage (92.3) of positive results was observed in Macaca mulatta with enteric diseases and in macaque dead of these diseases. The presence of C. jejuni in monkeys with diarrhea and the absence of pathogenic enterobacteria (Shigella, Salmonella, Yersinia) in feces probably attest to etiological relationship of acute enteric infections with Campylobacter.

The outbreak in Adler colony slightly differed from similar outbreak in Florida in 2014 by more marked hemorrhagic diathesis and articular changes in some monkeys caused by polyavitaminosis developed in the course of infection. Sensitive to infection were M. mulatta, M. fascicularis, Cercopithecus aethiops, P. hamadryas and anubis, and Cebus capucinus.

Carriership of agents of sexually transmitted diseases (Trichomonas, Chlamydia, Mycoplasma, Ureaplasma) is highly prevalent in healthy monkeys living in the Adler Breeding Center. The incidence of these microorganisms is appreciably higher in animals with gestoses and labor abnormalities in comparison with animals with normal genital function. Mixed infection caused by 2-4 agents is much more incident than monoinfection.

Screening of monkeys for anaerobic bacteria showed that the incidence of non-spore-forming anaerobes was 2.0-2.5 times higher than that of clostridia. The overwhelming majority of isolated anaerobes were saprophytes and opportunistic bacteria. The incidence of C. perfringens was higher in monkeys with enteric infections of obscure etiology and dead from these diseases than in healthy animals (48.2 vs. 36.8%).