2022
DOI: 10.1007/s11033-022-07922-z
|View full text |Cite
|
Sign up to set email alerts
|

A functional microRNA binding site variant in IL-23R gene in systemic lupus erythematosus and rheumatoid arthritis: is there any correlation?

Help me understand this report

Search citation statements

Order By: Relevance

Paper Sections

Select...
2

Citation Types

0
1
0
1

Year Published

2023
2023
2023
2023

Publication Types

Select...
2

Relationship

1
1

Authors

Journals

citations
Cited by 2 publications
(2 citation statements)
references
References 48 publications
0
1
0
1
Order By: Relevance
“…2 Basically, SLE emanates from uncontrolled auto-antibodies production against self-antigens comprising of DNA, nucleosomes, and histones and subsequent inflammation owing to the deposition of immune complexes. 3 The prevalence of SLE varies in different geographical areas ranging from 20 to 150 cases per 100,000 persons annually. 4 It has been reported that the prevalence of this disorder in Iran as a middle-east country is around 40 per 100,000 individuals.…”
Section: Introductionmentioning
confidence: 99%
“…2 Basically, SLE emanates from uncontrolled auto-antibodies production against self-antigens comprising of DNA, nucleosomes, and histones and subsequent inflammation owing to the deposition of immune complexes. 3 The prevalence of SLE varies in different geographical areas ranging from 20 to 150 cases per 100,000 persons annually. 4 It has been reported that the prevalence of this disorder in Iran as a middle-east country is around 40 per 100,000 individuals.…”
Section: Introductionmentioning
confidence: 99%
“…El análisis de cómo estos factores desencadenan una respuesta autoinmunitaria, relacionada con el inicio de la artritis reumatoide, ha derivado en un modelo preclínico que involucra al fenotipo de artritis reumatoide seropositiva, caracterizada por la elevación de biomarcadores, como el factor reumatoide y/o anticuerpos anti-péptido cíclico citrulinado, en fases previas a la enfermedad. Así, se sugiere que la interacción de los genes y el exposoma [11] [12] podrían interactuar en un período previo a la aparición de los primeros síntomas articulares. No obstante, este modelo no es equiparable para los fenotipos seronegativos [13].…”
Section: Introductionunclassified