Anorexia Nervosa and Pituitary Cachexia

Bruckner, William J.; Wies, Carl H.; Lavietes, Paul H.

doi:10.1097/00000441-193811000-00007

Cited by 22 publications

(6 citation statements)

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“…McCuUagh & Tupper (1940) and Escamilla & Lisser (1942) found that only I in 6 of these patients actually had Simmonds disease. The credit for differentiating between anorexia nervosa and Simmonds disease belongs to Bruckner et al (1938) who reiterated the psychological origin ofthe disease.…”

Section: Introductionmentioning

confidence: 99%

Anorexia nervosa: aspects of care and treatment

Armstrong-Esther

1977

Journal of Advanced Nursing

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The literature concerned with anorexia nervosa is reviewed. The picture ofa distinct, psychogenic syndrome emerges with a phobia about weight gain and preoccupation with eating. The illness occurs in young people and may take an obsessional or hysterical form. Treatment, in the first instance, consists of restoring calorific deficiency and body weight and attempting to prevent relapses. In this the nurse has a major role to play in supportive psychotherapy.

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Section: Introductionmentioning

confidence: 99%

Anorexia nervosa: aspects of care and treatment

Armstrong-Esther

1977

Journal of Advanced Nursing

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“…Bliss & Branch (1960) measured blood sugar in 5 cases of anorexia nervosa and found values between 2.6 and 4.6 mmol/l, while Bartels (1946) found only one of 7 cases to have a fasting blood sugar (3.1 mmol/l) below 3.9 mmol/l. One patient previously reported (Bruckner et al 1938) did have a fasting blood sugar as low as 1.4mmol/I but she, in common with the other cases, had no symptoms of hypoglycaemia.…”

Section: Discussionmentioning

confidence: 78%

Anorexia Nervosa Presenting as Reversible Hypoglycaemic Coma

Zalin

Lant

1984

J R Soc Med

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“…Previous studies have described severe hypoglycemia in anorexia nervosa as having no symptoms 44–46. Hypoglycemic coma is an unusual complication in anorexia nervosa, which has been described in some cases reflecting severe malnutrition and indicating a poor and possibly fatal prognosis 47,48.…”

Section: Resultsmentioning

confidence: 99%

Sudden death in eating disorders

Jáuregui¹,

Jáuregui‐Lobera²

2012

VHRM

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Eating disorders are usually associated with an increased risk of premature death with a wide range of rates and causes of mortality. “Sudden death” has been defined as the abrupt and unexpected occurrence of fatality for which no satisfactory explanation of the cause can be ascertained. In many cases of sudden death, autopsies do not clarify the main cause. Cardiovascular complications are usually involved in these deaths. The purpose of this review was to report an update of the existing literature data on the main findings with respect to sudden death in eating disorders by means of a search conducted in PubMed. The most relevant conclusion of this review seems to be that the main causes of sudden death in eating disorders are those related to cardiovascular complications. The predictive value of the increased QT interval dispersion as a marker of sudden acute ventricular arrhythmia and death has been demonstrated. Eating disorder patients with severe cardiovascular symptoms should be hospitalized. In general, with respect to sudden death in eating disorders, some findings (eg, long-term eating disorders, chronic hypokalemia, chronically low plasma albumin, and QT intervals >600 milliseconds) must be taken into account, and it must be highlighted that during refeeding, the adverse effects of hypophosphatemia include cardiac failure. Monitoring vital signs and performing electrocardiograms and serial measurements of plasma potassium are relevant during the treatment of eating disorder patients.

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Anorexia Nervosa and Pituitary Cachexia

Cited by 22 publications

References 0 publications

Anorexia nervosa: aspects of care and treatment

Anorexia nervosa: aspects of care and treatment

Anorexia Nervosa Presenting as Reversible Hypoglycaemic Coma

Sudden death in eating disorders

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