2008
DOI: 10.1016/s0072-9752(07)88030-4
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Chapter 30 The neuropsychology of vascular dementia

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Cited by 7 publications
(3 citation statements)
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“…VaD refers to a cumulative decline in cognitive functioning secondary to multiple or strategically placed infarctions, ischemic injury, or hemorrhagic lesions (for review, see Wetzel and Kramer 2008). Research diagnostic criteria for VaD require that multiple cognitive deficits occur in the presence of focal neurological signs and symptoms and/or laboratory (e.g., CT or MRI scan) evidence of cerebrovascular disease that is thought to be etiologically related to the cognitive impairment (Chui et al 1992;Roman et al 1993).…”
Section: Alzheimer Disease Versus Vascular Dementiamentioning
confidence: 99%
“…VaD refers to a cumulative decline in cognitive functioning secondary to multiple or strategically placed infarctions, ischemic injury, or hemorrhagic lesions (for review, see Wetzel and Kramer 2008). Research diagnostic criteria for VaD require that multiple cognitive deficits occur in the presence of focal neurological signs and symptoms and/or laboratory (e.g., CT or MRI scan) evidence of cerebrovascular disease that is thought to be etiologically related to the cognitive impairment (Chui et al 1992;Roman et al 1993).…”
Section: Alzheimer Disease Versus Vascular Dementiamentioning
confidence: 99%
“…Although the value of this heuristic has been disputed with regard to memory preservation (Reed et al, 2007), the clear impairment of executive process in small vessel disease is uncontroversial (Lamar et al, 2010). The selective vulnerability of frontal abilities to small vessel disease is likely due to the reliance of frontal lobe function on cerebral white matter pathways that are disproportionately impacted by disease of the small perforating arteries (Duering et al, 2011;Wetzel & Kramer, 2008). In its role of exerting mental control, inhibition, and motor planning, the frontal lobe requires dense interconnectivity with sensory and higher perceptual systems in the temporal, parietal, and occipital lobes.…”
Section: Neuropsychological Profiles Of Vascular Diseasementioning
confidence: 99%
“…Obviously each of these three mechanisms would have a different neuropsychological phenotype. For example, the fronto-basal-ganglionic disconnection is assumed to cause executive dysfunction, including attention deficit [46], and although there is some support for this assumption it has not been completely verified and as discussed above, whenever examined neuropsychologically there is considerable overlap between patients identified as having AD and those diagnosed as sub-cortical leucoencephalopathy [3,23]. Several reasons could account for this overlap, including comorbidity of AD in patients with vascular brain disease and vice versa, and the occurrence of vascular changes in patients with predominant AD.…”
Section: Introductionmentioning
confidence: 99%