Corneal Status in Primary Angle-closure Glaucoma With a History of Acute Attack

Chen, Mei Ju; Liu, Catherine Jui Ling; Cheng, Ching‐Yu; Lee, Shui Mei

doi:10.1097/ijg.0b013e3181fc800a

Cited by 48 publications

(35 citation statements)

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“…Unfortunately, we do not have data on the duration of the acute attack, but we speculate that early and rapid resolution of an acute attack reduces the risk of corneal endothelial damage in the long term. Our findings of the lack of correlation between ECD and disease duration and ocular biometric parameters corroborates the findings of Chen et al 29 in their study of acute PACG eyes. We also noted no correlation between ECD and glaucoma medications.…”

Section: Discussionsupporting

confidence: 93%

“…It is likely that changes in structure and function of corneal endothelial cells following an acute increase in IOP is transient and revert to pre-elevated IOP level once the acute crisis resolves. A study by Chen et al 29 found no significant differences in ECD of acute PACG eyes when compared with fellow PACG or chronic PACG eyes, while there was significantly lower ECD when compared with normal. Interestingly, a recent study on a rat model showed that an acute increase in IOP resulted in structural and functional damage of corneal endothelial cells, which gradually reversed when the acute rise in IOP resolved.…”

Section: Discussionmentioning

confidence: 91%

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Characteristics of the Corneal Endothelium Across the Primary Angle Closure Disease Spectrum

Verma

Nongpiur

Husain

et al. 2018

Invest. Ophthalmol. Vis. Sci.

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Section: Discussionsupporting

confidence: 93%

Section: Discussionmentioning

confidence: 91%

Characteristics of the Corneal Endothelium Across the Primary Angle Closure Disease Spectrum

Verma

Nongpiur

Husain

et al. 2018

Invest. Ophthalmol. Vis. Sci.

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“…; Chen et al. ). This loss was primarily affected by age and mean duration of attack (Malaise‐Stals et al.…”

Section: Discussionmentioning

confidence: 99%

“…; Chen et al. ). Laser interventions after acute attacks may therefore impose a higher risk of further endothelial damage.…”

Section: Discussionmentioning

confidence: 99%

Laser iridotomy and the corneal endothelium: a systemic review

Wang

Koh

Loon

2014

Acta Ophthalmologica

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ABSTRACT. This study aims to systemically review the effect of laser iridotomy on the corneal endothelium. Searches were performed for studies that either compared corneal endothelial cell density/count, corneal thickness and morphology pre-and postiridotomy, or evaluated the postiridotomy development of corneal decompensation. There were 26 eligible studies. Our review shows that the effect of laser iridotomy on the corneal endothelium has been investigated with varying results. Although it has been demonstrated to be a relatively safe procedure, there is still the potential long-term risk of corneal decompensation, for which a corneal transplantation may be indicated eventually. The longest interval between laser iridotomy and corneal decompensation reported was 8 years. Mechanisms proposed for endothelial damage include direct focal injury, thermal damage, mechanical shock waves, iris pigment dispersion, transient rise in intraocular pressure, inflammation, turbulent aqueous flow, time-dependent shear stress on endothelium, chronic breakdown of blood-aqueous barrier and damage from bubbles that settled onto the endothelium. Inherent risk factors identified were iridotrabecular contact, current or prior acute angle closure, pigmented irides, small iris-to-endothelium distance, pre-existing endothelial disease and diabetes. Intervention-related risk factors include laser type, delivery and quantity. The significance of the risk factors and their direct association with the development of corneal decompensation remain to be determined. Understanding these risk factors may allow physicians to counsel their patients better. They may offer opportunities for preventive strategies, allowing us to ensure that a procedure performed to prevent disease progression and visual loss does not cause further morbidity.

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“…The more dense the lens and the more shallow the anterior chamber, the higher the risk of the corneal endothelium being jeopardized from the prior acute angle closure. 21 Waiting for the development of elevated IOP would also contribute to the complexity of the cataract extraction. Laser gonioplasty would have been technically challenging due to unfavorable iris curvature.…”

Section: Discussionmentioning

confidence: 99%

Spectrum of Angle Closure, Uveal Effusion Syndrome, and Nanophthalmos

Areiter¹,

Neale²,

Johnson³

2016

Journal of Current Glaucoma Practice

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Nanophthalmos, uveal effusion syndrome, and acute angle closure glaucoma (ACG) can present as a continuum in a patient, as is described here. This patient's angle closure was thought to be caused by idiopathic uveal effusion syndrome, and while there are no generally accepted diagnosis criteria for nanophthalmos, our patient fulfilled the criteria as defined by Wu.10 To prevent development of further angle closure, the decision was made to do cataract extraction as opposed to medical management.How to cite this articleAreiter E, Neale M, Johnson SM. Spectrum of Angle Closure, Uveal Effusion Syndrome, and Nanophthalmos. J Curr Glaucoma Pract 2016;10(3):113-117.

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Corneal Status in Primary Angle-closure Glaucoma With a History of Acute Attack

Cited by 48 publications

References 0 publications

Characteristics of the Corneal Endothelium Across the Primary Angle Closure Disease Spectrum

Characteristics of the Corneal Endothelium Across the Primary Angle Closure Disease Spectrum

Laser iridotomy and the corneal endothelium: a systemic review

Spectrum of Angle Closure, Uveal Effusion Syndrome, and Nanophthalmos

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