1984
DOI: 10.1055/s-2007-1014733
|View full text |Cite
|
Sign up to set email alerts
|

Development of Individual Insulin Infusion Profiles for Open Loop Infusion Systems

Help me understand this report

Search citation statements

Order By: Relevance

Paper Sections

Select...
1

Citation Types

0
1
0

Year Published

1988
1988
1993
1993

Publication Types

Select...
3
2

Relationship

1
4

Authors

Journals

citations
Cited by 5 publications
(1 citation statement)
references
References 10 publications
0
1
0
Order By: Relevance
“…Finally, the late and augmented insulin levels ( fig. 5) in the high responders in the present study may also be caused by a delayed insulin response itself that is known to induce insulin resistance [34], Thus, the altered insulin se cretion in subjects with a 'high' postprandial triglyceride response may represent a very early stage or a variant of the so-called meta bolic syndrome in normal-weight subjects which was described by Haller [16] a few years ago and updated and precised by Reaven [17] in 1988. They hypothesized that insulin resistance and hyperinsulinemia might play a major role in the development of several atherogenic risk factors in obese pa tients [16][17][18], In summary, two postprandial phases after a lipid load can be differentiated: an early rather heterogenous response and a late more homogenous one which seem to be related.…”
Section: Discussionmentioning
confidence: 47%
“…Finally, the late and augmented insulin levels ( fig. 5) in the high responders in the present study may also be caused by a delayed insulin response itself that is known to induce insulin resistance [34], Thus, the altered insulin se cretion in subjects with a 'high' postprandial triglyceride response may represent a very early stage or a variant of the so-called meta bolic syndrome in normal-weight subjects which was described by Haller [16] a few years ago and updated and precised by Reaven [17] in 1988. They hypothesized that insulin resistance and hyperinsulinemia might play a major role in the development of several atherogenic risk factors in obese pa tients [16][17][18], In summary, two postprandial phases after a lipid load can be differentiated: an early rather heterogenous response and a late more homogenous one which seem to be related.…”
Section: Discussionmentioning
confidence: 47%